Friday, 14 January 2011 18:40

Selected Acute Physiological Outcomes

Andrew Steptoe and Tessa M. Pollard

The acute physiological adjustments recorded during the performance of problem-solving or psychomotor tasks in the laboratory include: raised heart rate and blood pressure; alterations in cardiac output and peripheral vascular resistance; increased muscle tension and electrodermal (sweat gland) activity; disturbances in breathing pattern; and modifications in gastrointestinal activity and immune function. The best studied neurohormonal responses are those of the catecholamines (adrenaline and noradrenaline) and cortisol. Noradrenaline is the primary transmitter released by the nerves of the sympathetic branch of the autonomic nervous system. Adrenaline is released from the adrenal medulla following stimulation of the sympathetic nervous system, while activation of the pituitary gland by higher centres in the brain results in the release of cortisol from the adrenal cortex. These hormones support autonomic activation during stress and are responsible for other acute changes, such as stimulation of the processes that govern blood clotting, and the release of stored energy supplies from adipose tissue. It is likely that these kinds of response will also be seen during job stress, but studies in which work conditions are simulated, or in which people are tested in their normal jobs, are required to demonstrate such effects.

A variety of methods is available to monitor these responses. Conventional psychophysiological techniques are used to assess autonomic responses to demanding tasks (Cacioppo and Tassinary 1990). Levels of stress hormones can be measured in the blood or urine, or in the case of cortisol, in the saliva. The sympathetic activity associated with challenge has also been documented by measures of noradrenaline spillover from nerve terminals, and by direct recording of sympathetic nervous activity with miniature electrodes. The parasympathetic or vagal branch of the autonomic nervous system typically responds to task performance with reduced activity, and this can, under certain circumstances, be indexed through recording heart rate variability or sinus arrhythmia. In recent years, power spectrum analysis of heart rate and blood pressure signals has revealed wave bands that are characteristically associated with sympathetic and parasympathetic activity. Measures of the power in these wavebands can be used to index autonomic balance, and have shown a shift towards the sympathetic branch at the expense of the parasympathetic branch during task performance.

Few laboratory assessments of acute physiological responses have simulated work conditions directly. However, dimensions of task demand and performance that are relevant to work have been investigated. For example, as the demands of externally paced work increase (through faster pace or more complex problem solving), there is a rise in adrenaline level, heart rate and blood pressure, a reduction in heart rate variability and an increase in muscle tension. In comparison with self-paced tasks performed at the same rate, external pacing results in greater blood pressure and heart rate increases (Steptoe et al. 1993). In general, personal control over potentially stressful stimuli reduces autonomic and neuroendocrine activation in comparison with uncontrollable situations, although the effort of maintaining control over the situation itself has its own physiological costs.

Frankenhaeuser (1991) has suggested that adrenaline levels are raised when a person is mentally aroused or performing a demanding task, and that cortisol levels are raised when an individual is distressed or unhappy. Applying these ideas to job stress, Frankenhaeuser has proposed that job demand is likely to lead to increased effort and thus to raise levels of adrenaline, while lack of job control is one of the main causes of distress at work and is therefore likely to stimulate raised cortisol levels. Studies comparing levels of these hormones in people doing their normal work with levels in the same people at leisure have shown that adrenaline is normally raised when people are at work. Effects for noradrenaline are inconsistent and may depend on the amount of physical activity that people carry out during work and leisure time. It has also been shown that adrenaline levels at work correlate positively with levels of job demand. In contrast, cortisol levels have not been shown typically to be raised in people at work, and it is yet to be demonstrated that cortisol levels vary according to the degree of job control. In the “Air Traffic Controller Health Change Study”, only a small proportion of workers produced consistent increases in cortisol as the objective workload became greater (Rose and Fogg 1993).

Thus only adrenaline among the stress hormones has been shown conclusively to rise in people at work, and to do so according to the level of demand they experience. There is evidence that levels of prolactin increase in response to stress while levels of testosterone decrease. However, studies of these hormones in people at work are very limited. Acute changes in the concentration of cholesterol in the blood have also been observed with increased workload, but the results are not consistent (Niaura, Stoney and Herbst 1992).

As far as cardiovascular variables are concerned, it has repeatedly been found that blood pressure is higher in men and women during work than either after work or during equivalent times of day spent at leisure. These effects have been observed both with self-monitored blood pressure and with automated portable (or ambulatory) monitoring instruments. Blood pressure is especially high during periods of increased work demand (Rose and Fogg 1993). It has also been found that blood pressure rises with emotional demands, for example, in studies of paramedics attending the scenes of accidents. However, it is often difficult to determine whether blood pressure fluctuations at work are due to psychological demands or to associated physical activity and changes in posture. The raised blood pressure recorded at work is especially pronounced among people reporting high job strain according to the Demand-Control model (Schnall et al. 1990).

Heart rate has not been shown to be consistently raised during work. Acute elevations of heart rate may nevertheless be elicited by disruption of work, for example with breakdown of equipment. Emergency workers such as fire-fighters exhibit extremely fast heart rates in response to alarm signals at work. On the other hand, high levels of social support at work are associated with reduced heart rates. Abnormalities of cardiac rhythm may also be elicited by stressful working conditions, but the pathological significance of such responses has not been established.

Gastrointestinal problems are commonly reported in studies of job stress (see “Gastrointestinal problems” below). Unfortunately, it is difficult to assess the physiological systems underlying gastrointestinal symptoms in the work setting. Acute mental stress has variable effects on gastric acid secretion, stimulating large increases in some individuals and reduced output in others. Shift workers have a particularly high prevalence of gastrointestinal problems, and it has been suggested that these may arise when diurnal rhythms in the central nervous system’s control of gastric acid secretion are disrupted. Anomalies of small bowel motility have been recorded using radiotelemetry in patients diagnosed with irritable bowel syndrome while they go about their everyday lives. Health complaints, including gastrointestinal symptoms, have been shown to co-vary with perceived workload, but it is not clear whether this reflects objective changes in physiological function or patterns of symptom perception and reporting.


Back

Friday, 14 January 2011 18:39

Ethnicity

Major changes are taking place within the workforces of many of the world’s leading industrial nations, with members of ethnic minority groups making up increasingly larger proportions. However, little of the occupational stress research has focused on ethnic minority populations. The changing demographics of the world’s workforce give clear notice that these populations can no longer be ignored. This article briefly addresses some of the major issues of occupational stress in ethnic minority populations with a focus on the United States. However, much of the discussion should be generalizable to other nations of the world.

Much of the occupational stress research either excludes ethnic minorities, includes too few to allow meaningful comparisons or generalizations to be made, or does not report enough information about the sample to determine racial or ethnic participation. Many studies fail to make distinctions among ethnic minorities, treating them as one homogeneous group, thus minimizing the differences in demographic characteristics, culture, language and socio-economic status which have been documented both between and within ethnic minority groups (Olmedo and Parron 1981).

In addition to the failure to address issues of ethnicity, by far the greater part of research does not examine class or gender differences, or class by race and gender interactions. Moreover, little is known about the cross-cultural utility of many of the assessment procedures. Documentation used in such procedures is not adequately translated nor is there demonstrated equivalency between the standardized English and other language versions. Even when the reliabilities appear to indicate equivalence across ethnic or cultural groups, there is uncertainty about which symptoms in the scale are elicited in a reliable fashion, that is, whether the phenomenology of a disorder is similar across groups (Roberts, Vernon and Rhoades 1989).

Many assessment instruments inadequately assess conditions within ethnic minority populations; consequently results are often suspect. For example, many stress scales are based on models of stress as a function of undesirable change or readjustment. However, many minority individuals experience stress in large part as a function of ongoing undesirable situations such as poverty, economic marginality, inadequate housing, unemployment, crime and discrimination. These chronic stressors are not usually reflected in many of the stress scales. Models which conceptualize stress as resulting from the interplay between both chronic and acute stressors, and various internal and external mediating factors, are more appropriate for assessing stress in ethnic minority and poor populations (Watts-Jones 1990).

A major stressor affecting ethnic minorities is the prejudice and discrimination they encounter as a result of their minority status in a given society (Martin 1987; James 1994). It is a well- established fact that minority individuals experience more prejudice and discrimination as a result of their ethnic status than do members of the majority. They also perceive greater discrimination and fewer opportunities for advancement as compared with whites (Galinsky, Bond and Friedman 1993). Workers who feel discriminated against or who feel that there are fewer chances for advancement for people of their ethnic group are more likely to feel “burned out” in their jobs, care less about working hard and doing their jobs well, feel less loyal to their employers, are less satisfied with their jobs, take less initiative, feel less committed to helping their employers succeed and plan to leave their current employers sooner (Galinsky, Bond and Friedman 1993). Moreover, perceived prejudice and discrimination are positively correlated with self-reported health problems and higher blood pressure levels (James 1994).

An important focus of occupational stress research has been the relationship between social support and stress. However, there has been little attention paid to this variable with respect to ethnic minority populations. The available research tends to show conflicting results. For example, Hispanic workers who reported higher levels of social support had less job-related tension and fewer reported health problems (Gutierres, Saenz and Green 1994); ethnic minority workers with lower levels of emotional support were more likely to experience job burn-out, health symptoms, episodic job stress, chronic job stress and frustration; this relationship was strongest for women and for management as opposed to non-management personnel (Ford 1985). James (1994), however, did not find a significant relationship between social support and health outcomes in a sample of African-American workers.

Most models of job satisfaction have been derived and tested using samples of white workers. When ethnic minority groups have been included, they have tended to be African-Americans, and potential effects due to ethnicity were often masked (Tuch and Martin 1991). Research that is available on African-American employees tends to yield significantly lower scores on overall job satisfaction in comparison to whites (Weaver 1978, 1980; Staines and Quinn 1979; Tuch and Martin 1991). Examining this difference, Tuch and Martin (1991) noted that the factors determining job satisfaction were basically the same but that African-Americans were less likely to have the situations that led to job satisfaction. More specifically, extrinsic rewards increase African-Americans’ job satisfaction, but African-Americans are disadvantaged relatively to whites on these variables. On the other hand, blue-collar incumbency and urban residence decrease job satisfaction for African-Americans but African-Americans are overrepresented in these areas. Wright, King and Berg (1985) found that organizational variables (i.e., job authority, qualifications for the position and a sense that advancement within the organization is possible) were the best predictors of job satisfaction in their sample of black female managers in keeping with previous research on primarily white samples.

Ethnic minority workers are more likely than their white counterparts to be in jobs with hazardous work conditions. Bullard and Wright (1986/1987) noted this propensity and indicated that the population differences in injuries are likely to be the result of racial and ethnic disparities in income, education, type of employment and other socio-economic factors correlated with exposure to hazards. One of the most likely reasons, they noted, was that occupational injuries are highly dependent on the job and industry category of the workers and ethnic minorities tend to work in more hazardous occupations.

Foreign workers who have entered the country illegally often experience special work stress and maltreatment. They often endure substandard and unsafe working conditions and accept less than minimum wages because of fear of being reported to the immigration authorities and they have few options for better employment. Most health and safety regulations, guidelines for use, and warnings are in English and many immigrants, illegal or otherwise, may not have a good understanding of written or spoken English (Sanchez 1990).

Some areas of research have almost totally ignored ethnic minority populations. For example, hundreds of studies have examined the relationship between Type A behaviour and occupational stress. White males constitute the most frequently studied groups with ethnic minority men and women almost totally excluded. Available research—e.g., a study by Adams et al. (1986), using a sample of college freshmen, and e.g., Gamble and Matteson (1992), investigating black workers—indicates the same positive relationship between Type A behaviour and self-reported stress as that found for white samples.

Similarly, little research on issues such as job control and work demands is available for ethnic minority workers, although these are central constructs in occupational stress theory. Available research tends to show that these are important constructs for ethnic minority workers as well. For example, African-American licensed practical nurses (LPNs) report significantly less decision authority and more dead-end jobs (and hazard exposures) than do white LPNs and this difference is not a function of educational differences (Marshall and Barnett 1991); the presence of low decision latitude in the face of high demands tends to be the pattern most characteristic of jobs with low socio-economic status, which are more likely to be held by ethnic minority workers (Waitzman and Smith 1994); and middle- and upper-level white men rate their jobs consistently higher than their ethnic minority (and female) peers on six work design factors (Fernandez 1981).

Thus, it appears that many research questions remain regarding ethnic minority populations in the occupational stress and health arena as regards ethnic minority populations. These questions will not be answered until ethnic minority workers are included in study samples and in the development and validation of investigatory instruments.


Back

Friday, 14 January 2011 18:27

Gender, Job Stress and Illness

Do job stressors affect men and women differently? This question has only recently been addressed in the job stress–illness literature. In fact, the word gender does not even appear in the index of the first edition of the Handbook of Stress (Goldberger and Breznitz 1982) nor does it appear in the indices of such major reference books as Job Stress and Blue Collar Work (Cooper and Smith 1985) and Job Control and Worker Health (Sauter, Hurrell and Cooper 1989). Moreover, in a 1992 review of moderator variables and interaction effects in the occupational stress literature, gender effects were not even mentioned (Holt 1992). One reason for this state of affairs lies in the history of occupational health and safety psychology, which in turn reflects the pervasive gender stereotyping in our culture. With the exception of reproductive health, when researchers have looked at physical health outcomes and physical injuries, they have generally studied men and variations in their work. When researchers have studied mental health outcomes, they have generally studied women and variations in their social roles.

As a result, the “available evidence” on the physical health impact of work has until recently been almost completely limited to men (Hall 1992). For example, attempts to identify correlates of coronary heart disease have been focused exclusively on men and on aspects of their work; researchers did not even inquire into their male subjects’ marital or parental roles (Rosenman et al. 1975). Indeed, few studies of the job stress–illness relationship in men include assessments of their marital and parental relationships (Caplan et al. 1975).

In contrast, concern about reproductive health, fertility and pregnancy focused primarily on women. Not surprisingly, “the research on reproductive effects of occupational exposures is far more extensive on females than on males” (Walsh and Kelleher 1987). With respect to psychological distress, attempts to specify the psychosocial correlates, in particular the stressors associated with balancing work and family demands, have centred heavily on women.

By reinforcing the notion of “separate spheres” for men and women, these conceptualizations and the research paradigms they generated prevented any examination of gender effects, thereby effectively controlling for the influence of gender. Extensive sex segregation in the workplace (Bergman 1986; Reskin and Hartman 1986) also acts as a control, precluding the study of gender as a moderator. If all men are employed in “men’s jobs” and all women are employed in “women’s jobs”, it would not be reasonable to ask about the moderating effect of gender on the job stress–illness relationship: job conditions and gender would be confounded. It is only when some women are employed in jobs that men occupy and when some men are employed in jobs that women occupy that the question is meaningful.

Controlling is one of three strategies for treating the effects of gender. The other two are ignoring these effects or analysing them (Hall 1991). Most investigations of health have either ignored or controlled for gender, thereby accounting for the dearth of references to gender as discussed above and for a body of research that reinforces stereotyped views about the role of gender in the job stress–illness relationship. These views portray women as essentially different from men in ways that render them less robust in the workplace, and portray men as comparatively unaffected by non-workplace experiences.

In spite of this beginning, the situation is already changing. Witness the publication in 1987 of Gender and Stress (Barnett, Biener and Baruch 1987), the first edited volume focusing specifically on the impact of gender at all points in the stress reaction. And the second edition of the Handbook of Stress (Barnett 1992) includes a chapter on gender effects. Indeed, current studies increasingly reflect the third strategy: analysing gender effects. This strategy holds great promise, but also has pitfalls. Operationally, it involves analysing data relating to males and females and estimating both the main and the interaction effects of gender. A significant main effect tells us that after controlling for the other predictors in the model, men and women differ with respect to the level of the outcome variable. Interaction-effects analyses concern differential reactivity, that is, does the relationship between a given stressor and a health outcome differ for women and men?

The main promise of this line of inquiry is to challenge stereotyped views of women and men. The main pitfall is that conclusions about gender difference can still be drawn erroneously. Because gender is confounded with many other variables in our society, these variables have to be taken into account before conclusions about gender can be inferred. For example, samples of employed men and women will undoubtedly differ with respect to a host of work and non-work variables that could reasonably affect health outcomes. Most important among these contextual variables are occupational prestige, salary, part-time versus full-time employment, marital status, education, employment status of spouse, overall work burdens and responsibility for care of younger and older dependants. In addition, evidence suggests the existence of gender differences in several personality, cognitive, behavioural and social system variables that are related to health outcomes. These include: sensation seeking; self-efficacy (feelings of competence); external locus of control; emotion-focused versus problem-focused coping strategies; use of social resources and social support; harmful acquired risks, such as smoking and alcohol abuse; protective behaviours, such as exercise, balanced diets and preventive health regimens; early medical intervention; and social power (Walsh, Sorensen and Leonard, in press). The better one can control these contextual variables, the closer one can get to understanding the effect of gender per se on the relationships of interest, and thereby to understanding whether it is gender or other, gender-related variables that are the effective moderators.

To illustrate, in one study (Karasek 1990) job changes among white-collar workers were less likely to be associated with negative health outcomes if the changes resulted in increased job control. This finding was true for men, not women. Further analyses indicated that job control and gender were confounded. For women, one of “the less aggressive [or powerful] groups in the labour market” (Karasek 1990), white-collar job changes often involved reduced control, whereas for men, such job changes often involved increased control. Thus, power, not gender, accounted for this interaction effect. Such analyses lead us to refine the question about moderator effects. Do men and women react differentially to workplace stressors because of their inherent (i.e., biological) nature or because of their different experiences?

Although only a few studies have examined gender interaction effects, most report that when appropriate controls are utilized, the relationship between job conditions and physical or mental health outcomes is not affected by gender. (Lowe and Northcott 1988 describe one such study). In other words, there is no evidence of an inherent difference in reactivity.

Findings from a random sample of full-time employed men and women in dual-earner couples illustrates this conclusion with respect to psychological distress. In a series of cross-sectional and longitudinal analyses, a matched pairs design was used that controlled for such individual-level variables as age, education, occupational prestige and marital-role quality, and for such couple-level variables as parental status, years married and household income (Barnett et al. 1993; Barnett et al. 1995; Barnett, Brennan and Marshall 1994). Positive experiences on the job were associated with low distress; insufficient skill discretion and overload were associated with high distress; experiences in the roles of partner and parent moderated the relationship between job experiences and distress; and change over time in skill discretion and overload were each associated with change over time in psychological distress. In no case was the effect of gender significant. In other words, the magnitude of these relationships was not affected by gender.

One important exception is tokenism (see, for example, Yoder 1991). Whereas “it is clear and undeniable that there is a considerable advantage in being a member of the male minority in any female profession” (Kadushin 1976), the opposite is not true. Women who are in minority in a male work situation experience a considerable disadvantage. Such a difference is readily understandable in the context of men’s and women’s relative power and status in our culture.

Overall, studies of physical health outcomes also fail to reveal significant gender interaction effects. It appears, for example, that characteristics of work activity are stronger determinants of safety than are attributes of workers, and that women in traditionally male occupations suffer the same types of injury with approximately the same frequency as their male counterparts. Moreover, poorly designed protective equipment, not any inherent incapacity on the part of women in relation to the work, is often to blame when women in male-dominated jobs experience more injuries (Walsh, Sorensen and Leonard, 1995).

Two caveats are in order. First, no one study controls for all the gender-related covariates. Therefore, any conclusions about “gender” effects must be tentative. Secondly, because controls vary from study to study, comparisons between studies are difficult.

As increasing numbers of women enter the labour force and occupy jobs similar to those occupied by men, both the opportunity and the need for analysing the effect of gender on the job stress–illness relationship also increase. In addition, future research needs to refine the conceptualization and measurement of the stress construct to include job stressors important to women; extend interaction effects analyses to studies previously restricted to male or female samples, for example, studies of reproductive health and of stresses due to non-workplace variables; and examine the interaction effects of race and class as well as the joint interaction effects of gender x race and gender x class.


Back

Friday, 14 January 2011 18:13

Social Support

During the mid-1970s public health practitioners, and in particular, epidemiologists “discovered” the concept of social support in their studies of causal relationships between stress, mortality and morbidity (Cassel 1974; Cobb 1976). In the past decade there has been an explosion in the literature relating the concept of social support to work-related stressors. By contrast, in psychology, social support as a concept had already been well integrated into clinical practice. Rogers’ (1942) client-centred therapy of unconditional positive regard is fundamentally a social support approach. Lindeman’s (1944) pioneering work on grief management identified the critical role of support in moderating the crisis of death loss. Caplin’s (1964) model of preventive community psychiatry (1964) elaborated on the importance of community and support groups.

Cassel (1976) adapted the concept of social support into public health theory as a way of explaining the differences in diseases that were thought to be stress-related. He was interested in understanding why some individuals appeared to be more resistant to stress than others. The idea of social support as a factor in disease causation was reasonable since, he noted, both people and animals who experienced stress in the company of “significant others” seemed to suffer fewer adverse consequences than those who were isolated. Cassel proposed that social support could act as a protective factor buffering an individual from the effects of stress.

Cobb (1976) expanded on the concept by noting that the mere presence of another person is not social support. He suggested that an exchange of “information” was needed. He established three categories for this exchange:

  • information leading the person to the belief that one is loved or cared for (emotional support)
  • information leading to the belief that one is esteemed and valued (esteem support)
  • information leading to the belief that one belongs to a network of mutual obligations and communication.

 

Cobb reported that those experiencing severe events without such social support were ten times more likely to come to be depressed and concluded that somehow intimate relations, or social support, was protective of the effects of stress reactions. He also proposed that social support operates throughout one’s life span, encompassing various life events such as unemployment, severe illness and bereavement. Cobb pointed out the great diversity of studies, samples, methods and outcomes as convincing evidence that social support is a common factor in modifying stress, but is, in itself, not a panacea for avoiding its effects.

According to Cobb, social support increases coping ability (environmental manipulation) and facilitates adaptation (self-change to improve the person-environment fit). He cautioned, however, that most research was focused on acute stressors and did not permit generalizations of the protective nature of social support for coping with the effects of chronic stressors or traumatic stress.

Over the intervening years since the publication of these seminal works, investigators have moved away from considering social support as a unitary concept, and have attempted to understand the components of social stress and social support.

Hirsh (1980) describes five possible elements of social support:

  • emotional support: care, comfort, love, affection, sympathy
  • encouragement: praise, compliments; the extent to which one feels inspired by the supporter to feel courage, hope or to prevail
  • advice: useful information to solve problems; the extent to which one feels informed
  • companionship: time spent with supporter; the extent to which one does not feel alone
  • tangible aid: practical resources, such as money or aid with chores; the extent to which one feels relieved of burdens. Another framework is used by House (1981), to discuss social support in the context of work-related stress:
  • emotional: empathy, caring, love, trust, esteem or demonstrations of concern
  • appraisal: information relevant to self-evaluation, feedback from others useful in self-affirmation
  • informational: suggestions, advice or information useful in problem-solving
  • instrumental: direct aid in the form of money, time or labour.

 

House felt that emotional support was the most important form of social support. In the workplace, the supportiveness of the supervisor was the most important element, followed by co-worker support. The structure and organization of the enterprise, as well as the specific jobs within it, could either enhance or inhibit potential for support. House found that greater task specialization and fragmentation of work leads to more isolated work roles and to decreased opportunities for support.

Pines’ (1983) study of burnout, which is a phenomenon discussed separately in this chapter, found that the availability of social support at work is negatively correlated with burnout. He identifies six different relevant aspects of social support which modify the burnout response. These include listening, encouragement, giving advice and, providing companionship and tangible aid.

As one may gather from the foregoing discussion in which the models proposed by several researchers have been described, while the field has attempted to specify the concept of social support, there is no clear consensus on the precise elements of the concept, although considerable overlap between models is evident.

Interaction between Stress and Social Support

Although the literature on stress and social support is quite extensive, there is still considerable debate as to the mechanisms by which stress and social support interact. A long-standing question is whether social support has a direct or indirect effect on health.

Main effect/Direct effect

Social support can have a direct or main effect by serving as a barrier to the effects of the stressor. A social support network may provide needed information or needed feedback in order to overcome the stressor. It may provide a person with the resources he or she needs to minimize the stress. An individual’s self-perception may also be influenced by group membership so as to provide self-confidence, a sense of mastery and skill and hence thereby a sense of control over the environment. This is relevant to Bandura’s (1986) theories of personal control as the mediator of stress effects. There appears to be a minimum threshold level of social contact required for good health, and increases in social support above the minimum are less important. If one considers social support as having a direct—or main—effect, then one can create an index by which to measure it (Cohen and Syme 1985; Gottlieb 1983).

Cohen and Syme (1985), however, also suggest that an alternative explanation to social support acting as a main effect is that it is the isolation, or lack of social support, which causes the ill health rather than the social support itself promoting better health. This is an unresolved issue. Gottlieb also raises the issue of what happens when the stress results in the loss of the social network itself, such as might occur during disasters, major accidents or loss of work. This effect has not yet been quantified.

Buffering/Indirect effect

The buffering hypothesis is that social support intervenes between the stressor and the stress response to reduce its effects. Buffering could change one’s perception of the stressor, thus diminishing its potency, or it could increases one’s coping skills. Social support from others may provide tangible aid in a crisis, or it may lead to suggestions that facilitate adaptive responses. Finally, social support may be the stress-modifying effect which calms the neuroendocrine system so that the person may be less reactive to the stressor.

Pines (1983) notes that the relevant aspect of social support may be in the sharing of a social reality. Gottlieb proposes that social support could offset self-recrimination and dispel notions that the individual is him or herself responsible for the problems. Interaction with a social support system can encourage the venting of fears and can assist re-establishing a meaningful social identity.

Additional Theoretical Issues

Research thus far has tended to treat social support as a static, given factor. While the issue of its change over time has been raised, little data exist on the time course of social support (Gottlieb 1983; Cohen and Syme 1985). Social support is, of course, fluid, just as the stressors that it affects. It varies as the individual passes through the stages of life. It can also change over the short-term experience of a particular stressful event (Wilcox 1981).

Such variability probably means that social support fulfils different functions during different developmental stages or during different phases of a crisis. For example at the onset of a crisis, informational support may be more essential than tangible aid. The source of support, its density and the length of time it is operative will also be in flux. The reciprocal relationship between stress and social support must be recognized. Some stressors themselves have a direct impact on available support. Death of a spouse, for example, usually reduces the extent of the network and may have serious consequences for the survivor (Goldberg et al. 1985).

Social support is not a magic bullet that reduces the impact of stress. Under certain conditions it may exacerbate or be the cause of stress. Wilcox (1981) noted that those with a denser kin network had more difficulties adjusting to divorce because their families were less likely to accept divorce as a solution to marital problems. The literature on addiction and family violence also shows possible severe negative effects of social networks. Indeed, as Pines and Aronson (1981) point out, much of professional mental health interventions are devoted to undoing destructive relationships, and to teaching interpersonal skills and to assisting people to recover from social rejection.

There are a large number of studies employing a variety of measures of the functional content of social support. These measures have a wide range of reliability and construct validity. Another methodological problem is that these analyses depend largely on the self-reports of those being studied. The responses will therefore of necessity be subjective and will cause one to wonder whether it is the actual event or level of social support that is important or whether it is the individual’s perception of support and outcomes that is more critical. If it is the perception that is critical, then it may be that some other, third variable, such as personality type, is affecting both stress and social support (Turner 1983). For example, a third factor, such as age or socio-economic status, may influence change in both social support and outcome, according to Dooley (1985). Solomon (1986) provides some evidence for this idea with a study of women who have been forced by financial constraints into involuntary interdependence on friends and kin. She found that such women opt out of these relationships as quickly as they are financially able to do so.

Thoits (1982) raises concerns about reverse causation. It may be, she points out, that certain disorders chase away friends and lead to loss of support. Studies by Peters-Golden (1982) and Maher (1982) on cancer victims and social support appear to be consistent with this proposition.

Social Support and Work Stress

Studies on the relationship between social support and work stress indicate that successful coping is related to the effective use of support systems (Cohen and Ahearn 1980). Successful coping activities have emphasized the use of both formal and informal social support in dealing with work stress. Laid-off workers, for example, are advised to actively seek support to provide informational, emotional and tangible support. There have been relatively few evaluations of the effectiveness of such interventions. It appears, however, that formal support is only effective in the short term and informal systems are necessary for longer-term coping. Attempts to provide institutional formal social support can create negative outcomes, since the anger and rage about layoff or bankruptcy, for example, may be displaced to those who provide the social support. Prolonged reliance on social support may create a sense of dependency and lowered self- esteem.

In some occupations, such as seafarers, fire-fighters or staff in remote locations such as on oil rigs, there is a consistent, long-term, highly defined social network which can be compared to a family or kin system. Given the necessity for small work groups and joint efforts, it is natural that a strong sense of social cohesion and support develops among workers. The sometimes hazardous nature of the work requires that workers develop mutual respect, trust and confidence. Strong bonds and interdependence are created when people are dependent on each other for their survival and well-being.

Further research on the nature of social support during routine periods, as well as downsizing or major organizational change, is necessary to further define this factor. For example, when an employee is promoted to a supervisory position, he or she normally must distance him or herself from the other members of the work group. Does this make a difference in the day-to-day levels of social support he or she receives or requires? Does the source of support shift to other supervisors or to the family or somewhere else? Do those in positions of responsibility or authority experience different work stressors? Do these individuals require different types, sources or functions of social support?

If the target of the group-based interventions is also changing the functions of social support or the nature of the network, does this provide a preventive effect in future stressful events?

What will be the effect of growing numbers of women in these occupations? Does their presence change the nature and functions of support for all or does each sex require different levels or types of support?

The workplace presents a unique opportunity to study the intricate web of social support. As a closed subculture, it provides a natural experimental setting for research into the role of social support, social networks and their interrelationships with acute, cumulative and traumatic stress.


Back

Friday, 14 January 2011 18:11

Coping Styles

Coping has been defined as “efforts to reduce the negative impacts of stress on individual well-being” (Edwards 1988). Coping, like the experience of work stress itself, is a complex, dynamic process. Coping efforts are triggered by the appraisal of situations as threatening, harmful or anxiety producing (i.e., by the experience of stress). Coping is an individual difference variable that moderates the stress-outcome relationship.

Coping styles encompass trait-like combinations of thoughts, beliefs and behaviours that result from the experience of stress and may be expressed independently of the type of stressor. A coping style is a dispositional variable. Coping styles are fairly stable over time and situations and are influenced by personality traits, but are different from them. The distinction between the two is one of generality or level of abstraction. Examples of such styles, expressed in broad terms, include: monitor-blunter (Miller 1979) and repressor-sensitizer (Houston and Hodges 1970). Individual differences in personality, age, experience, gender, intellectual ability and cognitive style affect the way an individual copes with stress. Coping styles are the result of both prior experience and previous learning.

Shanan (1967) offered an early perspective on what he termed an adaptive coping style. This “response set” was characterized by four ingredients: the availability of energy directly focused on potential sources of the difficulty; a clear distinction between events internal and external to the person; confronting rather than avoiding external difficulties; and balancing external demands with needs of the self. Antonovsky (1987) similarly suggests that, to be effective, the individual person must be motivated to cope, have clarified the nature and dimensions of the problem and the reality in which it exists, and then selected the most appropriate resources for the problem at hand.

The most common typology of coping style (Lazarus and Folkman 1984) includes problem-focused coping (which includes information seeking and problem solving) and emotion-focused coping (which involves expressing emotion and regulating emotions). These two factors are sometimes complemented by a third factor, appraisal-focused coping (whose components include denial, acceptance, social comparison, redefinition and logical analysis).

Moos and Billings (1982) distinguish among the following coping styles:

  • Active-cognitive. The person tries to manage their appraisal of the stressful situation.
  • Active-behavioural. This style involves behaviour dealing directly with the stressful situations.
  • Avoidance. The person avoids confronting the problem.

 

Greenglass (1993) has recently proposed a coping style termed social coping, which integrates social and interpersonal factors with cognitive factors. Her research showed significant relationships between various kinds of social support and coping forms (e.g., problem-focused and emotion-focused). Women, generally possessing relatively greater interpersonal competence, were found to make greater use of social coping.

In addition, it may be possible to link another approach to coping, termed preventive coping, with a large body of previously separate writing dealing with healthy lifestyles (Roskies 1991). Wong and Reker (1984) suggest that a preventive coping style is aimed at promoting one’s well-being and reducing the likelihood of future problems. Preventive coping includes such activities as physical exercise and relaxation, as well as the development of appropriate sleeping and eating habits, and planning, time management and social support skills.

Another coping style, which has been described as a broad aspect of personality (Watson and Clark 1984), involves the concepts of negative affectivity (NA) and positive affectivity (PA). People with high NA accentuate the negative in evaluating themselves, other people and their environment in general and reflect higher levels of distress. Those with high PA focus on the positives in evaluating themselves, other people and their world in general. People with high PA report lower levels of distress.

These two dispositions can affect a person’s perceptions of the number and magnitude of potential stressors as well as his or her coping responses (i.e., one’s perceptions of the resources that one has available, as well as the actual coping strategies that are used). Thus, those with high NA will report fewer resources available and are more likely to use ineffective (defeatist) strategies (such as releasing emotions, avoidance and disengagement in coping) and less likely to use more effective strategies (such as direct action and cognitive reframing). Individuals with high PA would be more confident in their coping resources and use more productive coping strategies.

Antonovsky’s (1979; 1987) sense of coherence (SOC) concept overlaps considerably with PA. He defines SOC as a generalized view of the world as meaningful and comprehensible. This orientation allows the person to first focus on the specific situation and then to act on the problem and the emotions associated with the problem. High SOC individuals have the motivation and the cognitive resources to engage in these sorts of behaviours likely to resolve the problem. In addition, high SOC individuals are more likely to realize the importance of emotions, more likely to experience particular emotions and to regulate them, and more likely to take responsibility for their circumstances instead of blaming others or projecting their perceptions upon them. Considerable research has since supplied support for Antonovsky’s thesis.

Coping styles can be described with reference to dimensions of complexity and flexibility (Lazarus and Folkman 1984). People using a variety of strategies exhibit a complex style; those preferring a single strategy exhibit a single style. Those who use the same strategy in all situations exhibit a rigid style; those who use different strategies in the same, or different, situations exhibit a flexible style. A flexible style has been shown to be more effective than a rigid style.

Coping styles are typically measured by using self-reported questionnaires or by asking individuals, in an open-ended way, how they coped with a particular stressor. The questionnaire developed by Lazarus and Folkman (1984), the “Ways of Coping Checklist”, is the most widely used measure of problem-focused and emotion-focused coping. Dewe (1989), on the other hand, has frequently used individuals’ descriptions of their own coping initiatives in his research on coping styles.

There are a variety of practical interventions that may be implemented with regard to coping styles. Most often, intervention consists of education and training in which individuals are presented with information, sometimes coupled with self-assessment exercises that enable them to examine their own preferred coping style as well as other varieties of coping styles and their potential usefulness. Such information is typically well received by the persons to whom the intervention is directed, but the demonstrated usefulness of such information in helping them cope with real life stressors is lacking. In fact, the few studies that considered individual coping (Shinn et al. 1984; Ganster et al. 1982) have reported limited practical value in such education, particularly when a follow-up has been undertaken (Murphy 1988).

Matteson and Ivancevich (1987) outline a study dealing with coping styles as part of a longer programme of stress management training. Improvements in three coping skills are addressed: cognitive, interpersonal and problem solving. Coping skills are classified as problem-focused or emotion-focused. Problem-focused skills include problem solving, time management, communication and social skills, assertiveness, lifestyle changes and direct actions to change environmental demands. Emotion-focused skills are designed to relieve distress and foster emotion regulation. These include denial, expressing feelings and relaxation.

The preparation of this article was supported in part by the Faculty of Administrative Studies, York University.


Back

Friday, 14 January 2011 18:01

Locus of Control

Locus of control (LOC) refers to a personality trait reflecting the generalized belief that either events in life are controlled by one’s own actions (an internal LOC) or by outside influences (an external LOC). Those with an internal LOC believe that they can exert control over life events and circumstances, including the associated reinforcements, that is, those outcomes which are perceived to reward one’s behaviours and attitudes. In contrast, those with an external LOC believe they have little control over life events and circumstances, and attribute reinforcements to powerful others or to luck.

The construct of locus of control emerged from Rotter’s (1954) social learning theory. To measure LOC, Rotter (1966) developed the Internal-External (I-E) scale, which has been the instrument of choice in most research studies. However, research has questioned the unidimensionality of the I-E scale, with some authors suggesting that LOC has two dimensions (e.g., personal control and social system control), and others suggesting that LOC has three dimensions (personal efficacy, control ideology and political control). More recently developed scales to measure LOC are multidimensional, or assess LOC for specific domains, such as health or work (Hurrell and Murphy 1992).

One of the most consistent and widespread findings in the general research literature is the association between an external LOC and poor physical and mental health (Ganster and Fusilier 1989). A number of studies in occupational settings report similar findings: workers with an external LOC tended to report more burnout, job dissatisfaction, stress and lower self-esteem than those with an internal LOC (Kasl 1989). Recent evidence suggests that LOC moderates the relationship between role stressors (role ambiguity and role conflict) and symptoms of distress (Cvetanovski and Jex 1994; Spector and O’Connell 1994).

However, research linking LOC beliefs and ill health is difficult to interpret for several reasons (Kasl 1989). First, there may be conceptual overlap between the measures of health and locus of control scales. Secondly, a dispositional factor, like negative affectivity, may be present which is responsible for the relationship. For example, in the study by Spector and O’Connell (1994), LOC beliefs correlated more strongly with negative affectivity than with perceived autonomy at work, and did not correlate with physical health symptoms. Thirdly, the direction of causality is ambiguous; it is possible that the work experience may alter LOC beliefs. Finally, other studies have not found moderating effects of LOC on job stressors or health outcomes (Hurrell and Murphy 1992).

The question of how LOC moderates job stressor-health relationships has not been well researched. One proposed mechanism involves the use of more effective, problem-focused coping behaviour by those with an internal LOC. Those with an external LOC might use fewer problem-solving coping strategies because they believe that events in their lives are outside their control. There is evidence that people with an internal LOC utilize more task-centred coping behaviours and fewer emotion-centred coping behaviours than those with an external LOC (Hurrell and Murphy 1992). Other evidence indicates that in situations viewed as changeable, those with an internal LOC reported high levels of problem-solving coping and low levels of emotional suppression, whereas those with an external LOC showed the reverse pattern. It is important to bear in mind that many workplace stressors are not under the direct control of the worker, and that attempts to change uncontrollable stressors might actually increase stress symptoms (Hurrell and Murphy 1992).

A second mechanism whereby LOC could influence stressor-health relationships is via social support, another moderating factor of stress and health relationships. Fusilier, Ganster and Mays (1987) found that locus of control and social support jointly determined how workers responded to job stressors and Cummins (1989) found that social support buffered the effects of job stress, but only for those with an internal LOC and only when the support was work-related.

Although the topic of LOC is intriguing and has stimulated a great deal of research, there are serious methodological problems attaching to investigations in this area which need to be addressed. For example, the trait-like (unchanging) nature of LOC beliefs has been questioned by research which showed that people adopt a more external orientation with advancing age and after certain life experiences such as unemployment. Furthermore, LOC may be measuring worker perceptions of job control, instead of an enduring trait of the worker. Still other studies have suggested that LOC scales may not only measure beliefs about control, but also the tendency to use defensive manoeuvres, and to display anxiety or proneness to Type A behaviour (Hurrell and Murphy 1992).

Finally, there has been little research on the influence of LOC on vocational choice, and the reciprocal effects of LOC and job perceptions. Regarding the former, occupational differences in the proportion of “internals” and “externals” may be evidence that LOC influences vocational choice (Hurrell and Murphy 1992). On the other hand, such differences might reflect exposure to the job environment, just as the work environment is thought to be instrumental in the development of the Type A behaviour pattern. A final alternative is that occupational differences in LOC are be due to “drift”, that is the movement of workers into or out of certain occupations as a result of job dissatisfaction, health concerns or desire for advancement.

In summary, the research literature does not present a clear picture of the influence of LOC beliefs on job stressor or health relationships. Even where research has produced more or less consistent findings, the meaning of the relationship is obscured by confounding influences (Kasl 1989). Additional research is needed to determine the stability of the LOC construct and to identify the mechanisms or pathways through which LOC influences worker perceptions and mental and physical health. Components of the path should reflect the interaction of LOC with other traits of the worker, and the interaction of LOC beliefs with work environment factors, including reciprocal effects of the work environment and LOC beliefs. Future research should produce less ambiguous results if it incorporates measures of related individual traits (e.g., Type A behaviour or anxiety) and utilizes domain-specific measures of locus of control (e.g., work).


Back

Friday, 14 January 2011 17:58

Self-Esteem

Low self-esteem (SE) has long been studied as a determinant of psychological and physiological disorders (Beck 1967; Rosenberg 1965; Scherwitz, Berton and Leventhal 1978). Beginning in the 1980s, organizational researchers have investigated self-esteem’s moderating role in relationships between work stressors and individual outcomes. This reflects researchers’ growing interest in dispositions that seem either to protect or make a person more vulnerable to stressors.

Self-esteem can be defined as “the favorability of individuals’ characteristic self-evaluations” (Brockner 1988). Brockner (1983, 1988) has advanced the hypothesis that persons with low SE (low SEs) are generally more susceptible to environmental events than are high SEs. Brockner (1988) reviewed extensive evidence that this “plasticity hypothesis” explains a number of organizational processes. The most prominent research into this hypothesis has tested self-esteem’s moderating role in the relationship between role stressors (role conflict and role ambiguity) and health and affect. Role conflict (disagreement among one’s received roles) and role ambiguity (lack of clarity concerning the content of one’s role) are generated largely by events that are external to the individual, and therefore, according to the plasticity hypothesis, high SEs would be less vulnerable to them.

In a study of 206 nurses in a large southwestern US hospital, Mossholder, Bedeian and Armenakis (1981) found that self-reports of role ambiguity were negatively related to job satisfaction for low SEs but not for high SEs. Pierce et al. (1993) used an organization-based measure of self-esteem to test the plasticity hypothesis on 186 workers in a US utility company. Role ambiguity and role conflict were negatively related to satisfaction only among low SEs. Similar interactions with organization-based self-esteem were found for role overload, environmental support and supervisory support.

In the studies reviewed above, self-esteem was viewed as a proxy (or alternative measure) for self-appraisals of competence on the job. Ganster and Schaubroeck (1991a) speculated that the moderating role of self-esteem on role stressors’ effects was instead caused by low SEs’ lack of confidence in influencing their social environment, the result being weaker attempts at coping with these stressors. In a study of 157 US fire-fighters, they found that role conflict was positively related to somatic health complaints only among low SEs. There was no such interaction with role ambiguity.

In a separate analysis of the data on nurses’ reported in their earlier study (Mossholder, Bedeian and Armenakis 1981), these authors (1982) found that peer group interaction had a significantly more negative relationship to self-reported tension among low SEs than among high SEs. Likewise, low SEs reporting high peer-group interaction were less likely to wish to leave the organization than were high SEs reporting high peer-group interaction.

Several measures of self-esteem exist in the literature. Possibly the most often used of these is the ten-item instrument developed by Rosenberg (1965). This instrument was used in the Ganster and Schaubroeck (1991a) study. Mossholder and his colleagues (1981, 1982) used the self-confidence scale from Gough and Heilbrun’s (1965) Adjective Check List. The organization-based measure of self-esteem used by Pierce et al. (1993) was a ten-item instrument developed by Pierce et al. (1989).

The research findings suggest that health reports and satisfaction among low SEs can be improved either by reducing their role stressors or increasing their self-esteem. The organization development intervention of role clarification (dyadic supervisor-subordinate exchanges directed at clarifying the subordinate’s role and reconciling incompatible expectations), when combined with responsibility charting (clarifying and negotiating the roles of different departments), proved successful in a randomized field experiment at reducing role conflict and role ambiguity (Schaubroeck et al. 1993). It seems unlikely, however, that many organizations will be able and willing to undertake this rather extensive practice unless role stress is seen as particularly acute.

Brockner (1988) suggested a number of ways organizations can enhance employee self-esteem. Supervision practices are a major area in which organizations can improve. Performance appraisal feedback which focuses on behaviours rather than on traits, providing descriptive information with evaluative summations, and participatively developing plans for continuous improvement, is likely to have fewer adverse effects on employee self-esteem, and it may even enhance the self-esteem of some workers as they discover ways to improve their performance. Positive reinforcement of effective performance events is also critical. Training approaches such as mastery modelling (Wood and Bandura 1989) also ensure that positive efficacy perceptions are developed for each new task; these perceptions are the basis of organization-based self-esteem.


Back

Friday, 14 January 2011 17:49

Hardiness

The characteristic of hardiness is based in an existential theory of personality and is defined as a person’s basic stance towards his or his place in the world that simultaneously expresses commitment, control and readiness to respond to challenge (Kobasa 1979; Kobasa, Maddi and Kahn 1982). Commitment is the tendency to involve oneself in, rather than experience alienation from, whatever one is doing or encounters in life. Committed persons have a generalized sense of purpose that allows them to identify with and find meaningful the persons, events and things of their environment. Control is the tendency to think, feel and act as if one is influential, rather than helpless, in the face of the varied contingencies of life. Persons with control do not naïvely expect to determine all events and outcomes but rather perceive themselves as being able to make a difference in the world through their exercise of imagination, knowledge, skill and choice. Challenge is the tendency to believe that change rather than stability is normal in life and that changes are interesting incentives to growth rather than threats to security. So far from being reckless adventurers, persons with challenge are rather individuals with an openness to new experiences and a tolerance of ambiguity that enables them to be flexible in the face of change.

Conceived of as a reaction and corrective to a pessimistic bias in early stress research that emphasized persons’ vulnerability to stress, the basic hardiness hypothesis is that individuals characterized by high levels of the three interrelated orientations of commitment, control and challenge are more likely to remain healthy under stress than those individuals who are low in hardiness. The personality possessing hardiness is marked by a way of perceiving and responding to stressful life events that prevents or minimizes the strain that can follow stress and that, in turn, can lead to mental and physical illness.

The initial evidence for the hardiness construct was provided by retrospective and longitudinal studies of a large group of middle- and upper-level male executives employed by a Midwestern telephone company in the United States during the time of the divestiture of American Telephone and Telegraph (ATT). Executives were monitored through yearly questionnaires over a five-year period for stressful life experiences at work and at home, physical health changes, personality characteristics, a variety of other work factors, social support and health habits. The primary finding was that under conditions of highly stressful life events, executives scoring high on hardiness are significantly less likely to become physically ill than are executives scoring low on hardiness, an outcome that was documented through self-reports of physical symptoms and illnesses and validated by medical records based on yearly physical examinations. The initial work also demonstrated: (a) the effectiveness of hardiness combined with social support and exercise to protect mental as well as physical health; and (b) the independence of hardiness with respect to the frequency and severity of stressful life events, age, education, marital status and job level. Finally, the body of hardiness research initially assembled as a result of the study led to further research that showed the generalizability of the hardiness effect across a number of occupational groups, including non-executive telephone personnel, lawyers and US Army officers (Kobasa 1982).

Since those basic studies, the hardiness construct has been employed by many investigators working in a variety of occupational and other contexts and with a variety of research strategies ranging from controlled experiments to more qualitative field investigations (for reviews, see Maddi 1990; Orr and Westman 1990; Ouellette 1993). The majority of these studies have basically supported and expanded the original hardiness formulation, but there have also been disconfirmations of the moderating effect of hardiness and criticisms of the strategies selected for the measurement of hardiness (Funk and Houston 1987; Hull, Van Treuren and Virnelli 1987).

Emphasizing individuals’ ability to do well in the face of serious stressors, researchers have confirmed the positive role of hardiness among many groups including, in samples studied in the United States, bus drivers, military air-disaster workers, nurses working in a variety of settings, teachers, candidates in training for a number of different occupations, persons with chronic illness and Asian immigrants. Elsewhere, studies have been carried out among businessmen in Japan and trainees in the Israeli defence forces. Across these groups, one finds an association between hardiness and lower levels of either physical or mental symptoms, and, less frequently, a significant interaction between stress levels and hardiness that provides support for the buffering role of personality. In addition, results establish the effects of hardiness on non-health outcomes such as work performance and job satisfaction as well as on burnout. Another large body of work, most of it conducted with college-student samples, confirms the hypothesized mechanisms through which hardiness has its health-protective effects. These studies demonstrated the influence of hardiness upon the subjects’ appraisal of stress (Wiebe and Williams 1992). Also relevant to construct validity, a smaller number of studies have provided some evidence for the psychophysiological arousal correlates of hardiness and the relationship between hardiness and various preventive health behaviours.

Essentially all of the empirical support for a link between hardiness and health has relied upon data obtained through self-report questionnaires. Appearing most often in publications is the composite questionnaire used in the original prospective test of hardiness and abridged derivatives of that measure. Fitting the broad-based definition of hardiness as defined in the opening words of this article, the composite questionnaire contains items from a number of established personality instruments that include Rotter’s Internal-External Locus of Control Scale (Rotter, Seeman and Liverant 1962), Hahn’s California Life Goals Evaluation Schedules (Hahn 1966), Maddi’s Alienation versus Commitment Test (Maddi, Kobasa and Hoover 1979) and Jackson’s Personality Research Form (Jackson 1974). More recent efforts at questionnaire development have led to the development of the Personal Views Survey, or what Maddi (1990) calls the “Third Generation Hardiness Test”. This new questionnaire addresses many of the criticisms raised with respect to the original measure, such as the preponderance of negative items and the instability of hardiness factor structures. Furthermore, studies of working adults in both the United States and the United Kingdom have yielded promising reports as to the reliability and validity of the hardiness measure. Nonetheless, not all of the problems have been resolved. For example, some reports show low internal reliability for the challenge component of hardiness. Another pushes beyond the measurement issue to raise a conceptual concern about whether hardiness should always be seen as a unitary phenomenon rather than a multidimensional construct made up of separate components that may have relationships with health independently of each other in certain stressful situations. The challenge to future on researchers hardiness is to retain both the conceptual and human richness of the hardiness notion while increasing its empirical precision.

Although Maddi and Kobasa (1984) describe the childhood and family experiences that support the development of personality hardiness, they and many other hardiness researchers are committed to defining interventions to increase adults’ stress- resistance. From an existential perspective, personality is seen as something that one is constantly constructing, and a person’s social context, including his or her work environment, is seen as either supportive or debilitating as regards the maintenance of hardiness. Maddi (1987, 1990) has provided the most thorough depiction and rationale for hardiness intervention strategies. He outlines a combination of focusing, situational reconstruction, and compensatory self-improvement strategies that he has used successfully in small group sessions to enhance hardiness and decrease the negative physical and mental effects of stress in the workplace.


Back

Friday, 14 January 2011 17:44

Type A/B Behaviour Pattern

Definition

The Type A behaviour pattern is an observable set of behaviours or style of living characterized by extremes of hostility, competitiveness, hurry, impatience, restlessness, aggressiveness (sometimes stringently suppressed), explosiveness of speech, and a high state of alertness accompanied by muscular tension. People with strong Type A behaviour struggle against the pressure of time and the challenge of responsibility (Jenkins 1979). Type A is neither an external stressor nor a response of strain or discomfort. It is more like a style of coping. At the other end of this bipolar continuum, Type B persons are more relaxed, cooperative, steady in their pace of activity, and appear more satisfied with their daily lives and the people around them.

The Type A/B behavioural continuum was first conceptualized and labelled in 1959 by the cardiologists Dr. Meyer Friedman and Dr. Ray H. Rosenman. They identified Type A as being typical of their younger male patients with ischaemic heart disease (IHD).

The intensity and frequency of Type A behaviour increases as societies become more industrialized, competitive and hurried. Type A behaviour is more frequent in urban than rural areas, in managerial and sales occupations than among technical workers, skilled craftsmen or artists, and in businesswomen than in housewives.

Areas of Research

Type A behaviour has been studied as part of the fields of personality and social psychology, organizational and industrial psychology, psychophysiology, cardiovascular disease and occupational health.

Research relating to personality and social psychology has yielded considerable understanding of the Type A pattern as an important psychological construct. Persons scoring high on Type A measures behave in ways predicted by Type A theory. They are more impatient and aggressive in social situations and spend more time working and less in leisure. They react more strongly to frustration.

Research that incorporates the Type A concept into organizational and industrial psychology includes comparisons of different occupations as well as employees’ responses to job stress. Under conditions of equivalent external stress, Type A employees tend to report more physical and emotional strain than Type B employees. They also tend to move into high-demand jobs (Type A behavior 1990).

Pronounced increases in blood pressure, serum cholesterol and catecholamines in Type A persons were first reported by Rosenman and al. (1975) and have since been confirmed by many other investigators. The tenor of these findings is that Type A and Type B persons are usually quite similar in chronic or baseline levels of these physiological variables, but that environmental demands, challenges or frustrations create far larger reactions in Type A than Type B persons. The literature has been somewhat inconsistent, partly because the same challenge may not physiologically activitate men or women of different backgrounds. A preponderance of positive findings continues to be published (Contrada and Krantz 1988).

The history of Type A/B behaviour as a risk factor for ischeamic heart disease has followed a common historical trajectory: a trickle then a flow of positive findings, a trickle then a flow of negative findings, and now intense controversy (Review Panel on Coronary-Prone Behavior and Coronary Heart Disease 1981). Broad-scope literature searches now reveal a continuing mixture of positive associations and non-associations between Type A behaviour and IHD. The general trend of the findings is that Type A behaviour is more likely to be positively associated with a risk of IHD:

  1. in cross-sectional and case-control studies rather than prospective studies
  2. in studies of general populations and occupational groups rather than studies limited to persons with cardiovascular disease or who score high on other IHD risk factors
  3. in younger study groups (under age 60) rather than older populations
  4. in countries still in the process of industrialization or still at the peak of their economic development.

 

The Type A pattern is not “dead” as an IHD risk factor, but in the future must be studied with the expectation that it may convey greater IHD risk only in certain sub-populations and in selected social settings. Some studies suggest that hostility may be the most damaging component of Type A.

A newer development has been the study of Type A behaviour as a risk factor for injuries and mild and moderate illnesses both in occupational and student groups. It is rational to hypothesize that people who are hurried and aggressive will incur the most accidents at work, in sports and on the highway. This has been found to be empirically true (Elander, West and French 1993). It is less clear theoretically why mild acute illnesses in a full array of physiologic systems should occur more often to Type A than Type B persons, but this has been found in a few studies (e. g. Suls and Sanders 1988). At least in some groups, Type A was found to be associated with a higher risk of future mild episodes of emotional distress. Future research needs to address both the validity of these associations and the physical and psychological reasons behind them.

Methods of Measurement

The Type A/B behaviour pattern was first measured in research settings by the Structured Interview (SI). The SI is a carefully administered clinical interview in which about 25 questions are asked at different rates of speed and with different degrees of challenge or intrusiveness. Special training is necessary for an interviewer to be certified as competent both to administer and interpret the SI. Typically, interviews are tape-recorded to permit subsequent study by other judges to ensure reliability. In comparative studies among several measures of Type A behaviour, the SI seems to have greater validity for cardiovascular and psychophysiological studies than is found for self-report questionnaires, but little is known about its comparative validity in psychological and occupational studies because the SI is used much less frequently in these settings.

Self-Report Measures

The most common self-report instrument is the Jenkins Activity Survey (JAS), a self-report, computer-scored, multiple-choice questionnaire. It has been validated against the SI and against the criteria of current and future IHD, and has accumulated construct validity. Form C, a 52-item version of the JAS published in 1979 by the Psychological Corporation, is the most widely used. It has been translated into most of the languages of Europe and Asia. The JAS contains four scales: a general Type A scale, and factor-analytically derived scales for speed and impatience, job involvement and hard-driving competitiveness. A short form of the Type A scale (13 items) has been used in epidemiological studies by the World Health Organization.

The Framingham Type A Scale (FTAS) is a ten-item questionnaire shown to be a valid predictor of future IHD for both men and women in the Framingham Heart Study (USA). It has also been used internationally both in cardiovascular and psychological research. Factor analysis divides the FTAS into two factors, one of which correlates with other measures of Type A behaviour while the second correlates with measures of neuroticism and irritability.

The Bortner Rating Scale (BRS) is composed of fourteen items, each in the form of an analogue scale. Subsequent studies have performed item-analysis on the BRS and have achieved greater internal consistency or greater predictability by shortening the scale to 7 or 12 items. The BRS has been widely used in international translations. Additional Type A scales have been developed internationally, but these have mostly been used only for specific nationalities in whose language they were written.

Practical Interventions

Systematic efforts have been under way for at least two decades to help persons with intense Type A behaviour patterns to change them to more of a Type B style. Perhaps the largest of these efforts was in the Recurrent Coronary Prevention Project conducted in the San Francisco Bay area in the 1980s. Repeated follow-up over several years documented that changes were achieved in many people and also that the rate of recurrent myocardial infarction was reduced in persons receiving the Type A behaviour reduction efforts as opposed to those receiving only cardiovascular counselling (Thoreson and Powell 1992).

Intervention in the Type A behaviour pattern is difficult to accomplish successfully because this behavioural style has so many rewarding features, particularly in terms of career advancement and material gain. The programme itself must be carefully crafted according to effective psychological principles, and a group process approach appears to be more effective than individual counselling.


Back

Friday, 14 January 2011 17:34

Career Stages

Introduction

The career stage approach is one way to look at career development. The way in which a researcher approaches the issue of career stages is frequently based on Levinson’s life stage development model (Levinson 1986). According to this model, people grow through specific stages separated by transition periods. At each stage a new and crucial activity and psychological adjustment may be completed (Ornstein, Cron and Slocum 1989). In this way, defined career stages can be, and usually are, based on chronological age. The age ranges assigned for each stage have varied considerably between empirical studies, but usually the early career stage is considered to range from the ages of 20 to 34 years, the mid-career from 35 to 50 years and the late career from 50 to 65 years.

According to Super’s career development model (Super 1957; Ornstein, Cron and Slocum 1989) the four career stages are based on the qualitatively different psychological task of each stage. They can be based either on age or on organizational, positional or professional tenure. The same people can recycle several times through these stages in their work career. For example, according to the Career Concerns Inventory Adult Form, the actual career stage can be defined at an individual or group level. This instrument assesses an individual’s awareness of and concerns with various tasks of career development (Super, Zelkowitz and Thompson 1981). When tenure measures are used, the first two years are seen as a trial period. The establishment period from two to ten years means career advancement and growth. After ten years comes the maintenance period, which means holding on to the accomplishments achieved. The decline stage implies the development of one’s self-image independently of one’s career.

Because the theoretical bases of the definition of the career stages and the sorts of measure used in practice differ from one study to another, it is apparent that the results concerning the health- and job-relatedness of career development vary, too.

Career Stage as a Moderator of Work-Related Health and Well-Being

Most studies of career stage as a moderator between job characteristics and the health or well-being of employees deal with organizational commitment and its relation to job satisfaction or to behavioural outcomes such as performance, turnover and absenteeism (Cohen 1991). The relationship between job characteristics and strain has also been studied. The moderating effect of career stage means statistically that the average correlation between measures of job characteristics and well-being varies from one career stage to another.

Work commitment usually increases from early career stages to later stages, although among salaried male professionals, job involvement was found to be lowest in the middle stage. In the early career stage, employees had a stronger need to leave the organization and to be relocated (Morrow and McElroy 1987). Among hospital staff, nurses’ measures of well-being were most strongly associated with career and affective-organizational commitment (i.e., emotional attachment to the organization). Continuance commitment (this is a function of perceived number of alternatives and degree of sacrifice) and normative commitment (loyalty to organization) increased with career stage (Reilly and Orsak 1991).

A meta-analysis was carried out of 41 samples dealing with the relationship between organizational commitment and outcomes indicating well-being. The samples were divided into different career stage groups according to two measures of career stage: age and tenure. Age as a career stage indicator significantly affected turnover and turnover intentions, while organizational tenure was related to job performance and absenteeism. Low organizational commitment was related to high turnover, especially in the early career stage, whereas low organizational commitment was related to high absenteeism and low job performance in the late career stage (Cohen 1991).

The relationship between work attitudes, for instance job satisfaction and work behaviour, has been found to be moderated by career stage to a considerable degree (e.g., Stumpf and Rabinowitz 1981). Among employees of public agencies, career stage measured with reference to organizational tenure was found to moderate the relationship between job satisfaction and job performance. Their relation was strongest in the first career stage. This was supported also in a study among sales personnel. Among academic teachers, the relationship between satisfaction and performance was found to be negative during the first two years of tenure.

Most studies of career stage have dealt with men. Even many early studies in the 1970s, in which the sex of the respondents was not reported, it is apparent that most of the subjects were men. Ornstein and Lynn (1990) tested how the career stage models of Levinson and Super described differences in the career attitudes and intentions among professional women. The results suggest that career stages based on age were related to organizational commitment, intention to leave the organization and a desire for promotion. These findings were, in general, similar to the ones found among men (Ornstein, Cron and Slocum 1989). However, no support was derived for the predictive value of career stages as defined on a psychological basis.

Studies of stress have generally either ignored age, and consequently career stage, in their study designs or treated it as a confounding factor and controlled its effects. Hurrell, McLaney and Murphy (1990) contrasted the effects of stress in mid-career to its effects in early and late career using age as a basis for their grouping of US postal workers. Perceived ill health was not related to job stressors in mid-career, but work pressure and underutilization of skills predicted it in early and late career. Work pressure was related also to somatic complaints in the early and late career group. Underutilization of abilities was more strongly related to job satisfaction and somatic complaints among mid-career workers. Social support had more influence on mental health than physical health, and this effect is more pronounced in mid-career than in early or late career stages. Because the data were taken from a cross sectional study, the authors mention that cohort explanation of the results might also be possible (Hurrell, McLaney and Murphy 1990).

When adult male and female workers were grouped according to age, the older workers more frequently reported overload and responsibility as stressors at work, whereas the younger workers cited insufficiency (e.g., not challenging work), boundary-spanning roles and physical environment stressors (Osipow, Doty and Spokane 1985). The older workers reported fewer of all kinds of strain symptoms: one reason for this may be that older people used more rational-cognitive, self-care and recreational coping skills, evidently learned during their careers, but selection that is based on symptoms during one’s career may also explain these differences. Alternatively it might reflect some self-selection, when people leave jobs that stress them excessively over time.

Among Finnish and US male managers, the relationship between job demands and control on the one hand, and psychosomatic symptoms on the other, was found in the studies to vary according to career stage (defined on the basis of age) (Hurrell and Lindström 1992, Lindström and Hurrell 1992). Among US managers, job demands and control had a significant effect on symptom reporting in the middle career stage, but not in the early and late stage, while among Finnish managers, the long weekly working hours and low job control increased stress symptoms in the early career stage, but not in the later stages. Differences between the two groups might be due to the differences in the two samples studied. The Finnish managers, being in the construction trades, had high workloads already in their early career stage, whereas US managers—these were public sector workers—had the highest workloads in their middle career stage.

To sum up the results of research on the moderating effects of career stage: early career stage means low organizational commitment related to turnover as well as job stressors related to perceived ill health and somatic complaints. In mid-career the results are conflicting: sometimes job satisfaction and performance are positively related, sometimes negatively. In mid-career, job demands and low control are related to frequent symptom reporting among some occupational groups. In late career, organizational commitment is correlated to low absenteeism and good performance. Findings on relations between job stressors and strain are inconsistent for the late career stage. There are some indications that more effective coping decreases work-related strain symptoms in late career.

Interventions

Practical interventions to help people to cope better with the specific demands of each career stage would be beneficial. Vocational counselling at the entry stage of one’s work life would be especially useful. Interventions for minimizing the negative impact of career plateauing are suggested because this can be either a time of frustration or an opportunity to face new challenges or to reappraise one’s life goals (Weiner, Remer and Remer 1992). Results of age-based health examinations in occupational health services have shown that job-related problems lowering working ability gradually increase and qualitatively change with age. In early and mid-career they are related to coping with work overload, but in later middle and late career they are gradually accompanied by declining psychological condition and physical health, facts that indicate the importance of early institutional intervention at an individual level (Lindström, Kaihilahti and Torstila 1988). Both in research and in practical interventions, mobility and turnover pattern should be taken into account, as well as the role played by one’s occupation (and situation within that occupation) in one’s career development.


Back

Page 118 of 122

" DISCLAIMER: The ILO does not take responsibility for content presented on this web portal that is presented in any language other than English, which is the language used for the initial production and peer-review of original content. Certain statistics have not been updated since the production of the 4th edition of the Encyclopaedia (1998)."

Contents

Preface
Part I. The Body
Part II. Health Care
Part III. Management & Policy
Part IV. Tools and Approaches
Part V. Psychosocial and Organizational Factors
Part VI. General Hazards
Part VII. The Environment
Part VIII. Accidents and Safety Management
Part IX. Chemicals
Part X. Industries Based on Biological Resources
Part XI. Industries Based on Natural Resources
Part XII. Chemical Industries
Part XIII. Manufacturing Industries
Part XIV. Textile and Apparel Industries
Part XV. Transport Industries
Part XVI. Construction
Part XVII. Services and Trade
Education and Training Services
Emergency and Security Services
Entertainment and the Arts
Health Care Facilities and Services
Ergonomics and Health Care
The Physical Environment and Health Care
Healthcare Workers and Infectious Diseases
Chemicals in the Health Care Environment
The Hospital Environment
Health Care Facilities and Services Resources
Hotels and Restaurants
Office and Retail Trades
Personal and Community Services
Public and Government Services
Transport Industry and Warehousing
Part XVIII. Guides

Health Care Facilities and Services References

Abdo, R and H Chriske. 1990. HAV-Infektionsrisiken im Krankenhaus, Altenheim und Kindertagesstätten. In Arbeitsmedizin im Gesundheitsdienst, Band 5, edited by F Hofmann and U Stössel. Stuttgart: Gentner Verlag.

Acton, W. 1848. On the advantages of caouchoue and gutta-percha in protecting the skin against the contagion of animal poisons. Lancet 12:588.

Ahlin, J. 1992. Interdisciplinary case studies in offices in Sweden. In Corporate Space and Architecture. Vol. 2. Paris: Ministére de l’équipment et du logement.

Akinori, H and O Hiroshi. 1985. Analysis of fatigue and health conditions among hospital nurses. J Science of Labour 61:517-578.

Allmeers, H, B Kirchner, H Huber, Z Chen, JW Walter, and X Baur. 1996. The latency period between exposure and the symptoms in allergy to natural latex: Suggestions for prevention. Dtsh Med Wochenschr 121 (25/26):823-828.

Alter, MJ. 1986. Susceptibility to varicella zoster virus among adults at high risk for exposure. Infec Contr Hosp Epid 7:448-451.

—. 1993. The detection, transmission, and outcome of hepatitis C infection. Infect Agents Dis 2:155-166.

Alter, MJ, HS Margolis, K Krawczynski, FN Judson, A Mares, WJ Alexander, PY Hu, JK Miller, MA Gerber, and RE Sampliner. 1992. The natural history of community-acquired hepatitis C in the United States. New Engl J Med 327:1899-1905.

American Conference of Governmental Industrial Hygienists (ACGIH). 1991. Documentation of the Threshold Limit Values and Biological Exposure Indices, 6th edition. Cincinnati, OH: ACGIH.

—. 1994. TLVs: Threshold Limit Values and Biological Exposure Indices for 1994-1995. Cincinnati, OH: ACGIH.

American Hospital Association (AHA). 1992. Implementing Safer Needle Practice. Chicago, IL: AHA.

American Institute of Architects. 1984. Determining Hospital Space Requirements. Washington, DC: American Institute of Architects Press.

American Institute of Architects Committee on Architecture for Health. 1987. Guidelines for Construction and Equipment of Hospital and Medical Facilities. Washington, DC: American Institute of Acrchitects Press.

American Society of Heating, Refrigerating and Air-conditioning Engineers (ASHRAE). 1987. Health facilities. In ASHRAE Handbook: Heating, Ventilating and Air-conditioning Systems and Applications. Atlanta, GA: ASHRAE.

Anon. 1996. New drugs for HIV infection. Medical Letter of Drugs and Therapeutics 38:37.

Axelsson, G, R Rylander, and I Molin. 1989. Outcome of pregnancy in relation to irregular and inconvenient work schedules. Brit J Ind Med 46:393-398.

Beatty, J SK Ahern, and R Katz. 1977. Sleep deprivation and the vigilance of anesthesiologists during simulated surgery. In Vigilance, edited by RR Mackie. New York: Plenum Press.

Beck-Friis, B, P Strang, and PO Sjöden. 1991. Work stress and job satisfaction in hospital-based home care. Journal of Palliative Care 7(3):15-21.

Benenson, AS (ed.). 1990. Control of Communicable Disease in Man, 15th edition. Washington, DC: American Public Health Association.

Bertold, H, F Hofmann, M Michaelis, D Neumann-Haefelin, G Steinert, and J Wölfle. 1994. Hepatitis C—Risiko für Beschäftigte im Gesundheitsdienst? In Arbeitsmedizin im Gesundheitsdienst, Band 7, edited by F Hofmann, G Reschauer, and U Stössel. Stuttgart: Gentner Verlag.

Bertram, DA. 1988. Characteristics of shifts and second-year resident performance in an emergency department. NY State J Med 88:10-14.

Berufsgenossenschaft für Gesundheitsdienst und Wohlfahrtspflege (BGW). 1994. Geschäftsbericht.

Bissel, L and R Jones. 1975. Disabled doctors ignored by peers. Presented at the American Medical Association Conference on the Imparied Physician, 11 April, San Francisco, CA.

Bitker, TE. 1976. Reaching out to the depressed physician. JAMA 236(15):1713-1716.

Blanchard, M, MM Cantel, M Faivre, J Girot, JP Ramette, D Thely, and M Estryn-Béhar. 1992. Incidence des rythmes biologiques sur le travail de nuit. In Ergonomie à l’hôpital, edited by M Estryn-Béhar, C Gadbois, and M Pottier. Toulouse: Edition Octares.

Blanpain, C and M Estryn-Béhar. 1990. Measures d’ambiance physique dans dix services hospitaliers. Performances 45:18-33.

Blaycock, B. 1995. Latex allergies: Overview, prevention and implications for nursing care. Ostomy Wound Manage 41(5):10-12,14-15.

Blazer, MJ, FJ Hickman, JJ Farmer, and DJ Brenner. 1980. Salmonella typhi: The laboratory as a reservoir of infection. Journal of Infectious Diseases 142:934-938.

Blow, RJ and MIV Jayson. 1988. Back pain. In Fitness for Work: The Medical Approach, edited by FC Edwards, RL McCallum, and PJ Taylor. Oxford: Oxford University Press.

Boehm, G and E Bollinger. 1990. Significance of environmental factors on the tolerated enteral feeding volumes for patients in neonatal intensive care units. Kinderarzliche Praxis 58(6):275-279.

Bongers, P, RD Winter, MAJ Kompier, and VV Hildebrandt. 1992. Psychosocial Factors at Work and Musculoskeletal Diseases. Review of the literature. Leiden, Netherlands: TNO.

Bouhnik, C, M Estryn-Béhar, B Kapitaniak, M Rocher, and P Pereau. 1989. Le roulage dans les établissements de soins. Document pour le médecin du travail. INRS 39:243-252.

Boulard, R. 1993. Les indices de santé mentale du personnel infirmier: l’impact de la charge de travail, de l’autonomie et du soutien social. In La psychologie du travail à l’aube du XXI° siècle. Actes du 7° Congrès de psychologie du travail de langue française. Issy-les-Moulineaux: Editions EAP.

Breakwell, GM. 1989. Facing Physical Violence. London: British Psychological Society.

Bruce, DL and MJ Bach. 1976. Effects of Trace Concentrations of Anesthetic Gases on Behavioral Performance of Operating Room Personnel. DHEW (NIOSH) Publication No. 76-169. Cincinnati, OH: NIOSH.

Bruce, DL, KA Eide, HW Linde, and JE Eckenhoff. 1968. Causes of death among anesthesiologists: A 20 years survey. Anesthesiology 29:565-569.

Bruce, DL, KA Eide, NJ Smith, F Seltzer, and MH Dykes. 1974. A prospective survey of anesthesiologists’ mortality, 1967-1974. Anesthesiology 41:71-74.

Burhill, D, DA Enarson, EA Allen, and S Grzybowski. 1985. Tuberculosis in female nurses in British Columbia. Can Med Assoc J 132:137.

Burke, FJ, MA Wilson, and JF McCord. 1995. Allergy to latex gloves in clinical practice: Case reports. Quintessence Int 26(12):859-863.

Buring, JE, CH Hennekens, SL Mayrent, B Rosner, ER Greenberg, and T Colton. 1985. Health experiences of operating room personnel. Anesthesiology 62: 325-330.

Burton, R. 1990. St. Mary’s Hospital, Isle of Wight: A suitable background for caring. Brit Med J 301:1423-1425.

Büssing, A. 1993. Stress and burnout in nursing: Studies in different work structures and work schedules. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Cabal, C, D Faucon, H Delbart, F Cabal, and G Malot. 1986. Construction d’une blanchisserie industrielle aux CHU de Saint-Etienne. Arch Mal Prof 48(5):393-394.

Callan, JR, RT Kelly, ML Quinn, JW Gwynne, RA Moore, FA Muckler, J Kasumovic, WM Saunders, RP Lepage, E Chin, I Schoenfeld, and DI Serig. 1995. Human Factors Evaluation of Remote Afterloading Brachytherapy. NUREG/CR-6125. Vol. 1. Washington, DC: Nuclear Regulatory Commission

Cammock, R. 1981. Primary Health Care Buildings: Briefing and Design Guide for Architects and Their Clients. London: Architectural Press.

Cardo, D, P Srivastava, C Ciesielski, R Marcus, P McKibben, D Culver, and D Bell. 1995. Case-control study of HIV seroconversion in health care workers after percutaneous exposure to HIV-infected blood (abstract). Infect Control Hosp Epidemiol 16 suppl:20.

Carillo, T, C Blanco, J Quiralte, R Castillo, M Cuevas, and F Rodriguez de Castro. 1995. Prevalence of latex allergy among greenhouse workers. J Allergy Clin Immunol 96(5/1):699-701.

Catananti, C and A Cambieri. 1990. Igiene e Tecnica Ospedaliera (Hospital Hygiene and Organization). Roma: II Pensiero Scientifico Editore.

Catananti, C, G Damiani, G Capelli, and G Manara. 1993. Building design and selection of materials and furnishings in the hospital: A review of international guidelines. In Indoor Air ’93, Proceedings of the 6th International Conference on Indoor Air Quality and Climate 2:641-646.

Catananti, C, G Capelli, G Damiani, M Volpe, and GC Vanini. 1994. Multiple criteria evaluation in planning selection of materials for health care facilities. Preliminary identification of criteria and variables. In Healthy Buildings ’94, Proceedings of the 3rd International Conference 1:103-108.

Cats-Baril, WL and JW Frymoyer. 1991. The economics of spinal disorders. In The Adult Spine, edited by JW Frymoyer. New York: Raven Press.

Centers for Disease Control (CDC). 1982. Acquired immunodeficiency syndrome (AIDS): Precautions for clinical laboratory staffs. Morb Mortal Weekly Rep 31:577-580.

—. 1983. Acquired immunodeficiency syndrome (AIDS): Precautions for health-care workers and allied professionals. Morb Mortal Weekly Rep 32:450-451.

—. 1987a. Human immunodeficiency virus infection in health-care workers exposed to blood of infected patients. Morb Mortal Weekly Rep 36:285-289.

—. 1987b. Recommendations for prevention of HIV transmission in health-care settings. Morb Mortal Weekly Rep 36 suppl 2:3S-18S.

—. 1988a. Universal precautions for prevention of transmission of human immunodeficiency virus, hepatitis B virus, and other bloodborne pathogens in health-care settings. Morb Mortal Weekly Rep 37:377-382,387-388.

—. 1988b. Guidelines for prevention of transmission of human immunodeficiency virus and hepatitis B virus to health-care and public-safety workers. Morb Mortal Weekly Rep 37 suppl 6:1-37.

—. 1989. Guidelines for prevention of transmission of human immunodeficiency virus and hepatitis B virus to health-care and public-safety workers. Morb Mortal Weekly Rep 38 suppl 6.

—. 1990. Public Health Service statement on management of occupational exposure to human immunodeficiency virus, including considerations regarding post-exposure use. Morb Mortal Weekly Rep 39 (No. RR-1).

—. 1991a. Hepatitis B virus: A comprehensive strategy for eliminating transmission in the United States through universal childhood vaccination: Recommendations of the Immunization Practices Advisory Committee (ACIP). Morb Mortal Weekly Rep 40 (No. RR-13).

—. 1991b. Recommendations for preventing transmission of human immunodeficiency virus and hepatitis B virus to patients during exposure-prone invasive procedures. Morb Mortal Weekly Rep 40 (No. RR-8).

—. 1993a. Recommended infection-control practices in dentistry. Morb Mortal Weekly Rep 42 (No. RR-8):1-12.

—. 1993b. Biosafety in Microbial and Biomedical Laboratories, 3rd edition. DHHS (CDC) Publication No. 93-8395. Atlanta, GA: CDC.

—. 1994a. HIV/AIDS Surveillance Report. Vol. 5(4). Atlanta, GA: CDC.

—. 1994b. HIV/AIDS Prevention Newsletter. Vol. 5(4). Atlanta, GA: CDC.

—. 1994c. Human immunodeficiency virus in household settings—United States. Morb Mortal Weekly Rep 43:347-356.

—. 1994d. HIV/AIDS Surveillance Report. Vol. 6(1). Atlanta, GA: CDC.

—. 1994e. Guidelines for preventing the transmission of Mycobacterium tuberculosis in health-care facilities. Morb Mortal Weekly Rep 43 (No. RR-13):5-50.

—. 1995. Case-control study of HIV seroconversion in health-care workers after percutaneous exposure to HIV-infected blood—France, United Kingdom, and United States. Morb Mortal Weekly Rep 44:929-933.

—. 1996a. HIV/AIDS Surveillance Report. Vol 8(2). Atlanta, GA: CDC.

—. 1996b. Update: Provisional Public Health Service recommendations for chemoprophylaxis after occupational exposure to HIV. Morb Mortal Weekly Rep 45:468-472.

Charney, W (ed.). 1994. Essentials of Modern Hospital Safety. Boca Raton, FL: Lewis Publishers.

Chou, T, D Weil, and P Arnmow. 1986. Prevalence of measles antibodies in hospital personnel. Infec Contr Hosp Epid 7:309-311.

Chriske, H and A Rossa. 1991. Hepatitis-C-Infektionsgefährdung des medizinischen Personals. In Arbeitsmedizin im Gesundheitsdienst, Band 5, edited by F Hofmann and U Stössel. Stuttgart: Gentner Verlag.

Clark, DC, E Salazar-Gruesco, P Grabler, J Fawcett. 1984. Predictors of depression during the first 6 months of internship. Am J Psychiatry 141:1095-1098.

Clemens, R, F Hofmann, H Berthold, and G Steinert. 1992. Prävalenz von Hepatitis, A, B und C bei Bewohern einer Einrichtung für geistig Behinderte. Sozialpädiatrie 14:357-364.

Cohen, EN. 1980. Anasthetic Exposure in the Workplace. Littleton, MA: PSG Publishing Co.

Cohen, EN, JW Bellville, and BW Brown, Jr. 1971. Anesthesia, pregnancy and miscarriage: A study of operating room nurses and anesthetists. Anesthesiology 35:343-347.

—. 1974. Occupational disease among operating room personnel: A national study. Anesthesiology 41:321-340.

—. 1975. A survey of anethestic health hazards among dentists. J Am Dent Assoc 90:1291-1296.

Commission of the European Communities. 1990. Recommendation of the Commission February 21, 1990, about Protection of People against Exposure to Radon in Indoor Environments. 90/143/Euratom (Italian Translation).

Cooper, JB. 1984. Toward prevention of anesthesic mishaps. International Anesthesiology Clinics 22:167-183.

Cooper, JB, RS Newbower, and RJ Kitz. 1984. An analysis of major errors and equipment failures in anesthesia management: Considerations for prevention and detection. Anesthesiology 60(1):34-42.

Costa, G, R Trinco, and G Schallenberg. 1992. Problems of thermal comfort in an operating room equipped with laminar air flow system In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar M, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Cristofari, M-F, M Estryn-Béhar, M Kaminski, and E Peigné. 1989. Le travail des femmes à l’hôpital. Informations Hospitalières 22/23:48-62.

Council of the European Communities. 1988. Directive December 21, 1988, to Draw Near the Laws of Member Countries about Building Products. 89/106/EEC (Italian translation).

de Chambost, M. 1994. Alarmes sonnantes, soignantes trébuchantes. Objectif soins 26:63-68.

de Keyser, V and AS Nyssen. 1993. Les erreurs humaines en anesthésies. Le Travail humain 56(2/3):243-266.

Decree of the President of Ministers Council. 1986. Directive to the Regions about Private Health Care Facilities Requirements. 27 June.

Dehlin, O, S Berg, GBS Andersson, and G Grimby. 1981. Effect of physical training and ergonomic counselling on the psychosocial perception of work and on the subjective assesment of low-back insuffuciency. Scand J Rehab 13:1-9.

Delaporte, MF, M Estryn-Béhar, G Brucker, E Peigne, and A Pelletier. 1990. Pathologie dermatologique et exercice professionnel en milieu hospitalier. Arch Mal Prof 51(2):83-88.

Denisco, RA, JN Drummond, and JS Gravenstein. 1987. The effect of fatigue on the performance of a simulated anesthetic monitoring task. J Clin Monit 3:22-24.

Devienne, A, D Léger, M Paillard, A Dômont. 1995. Troubles du sommeil et de la vigilance chez des généralistes de garde en région parisienne. Arch Mal Prof 56(5):407-409.

Donovan, R, PA Kurzman, and C Rotman. 1993. Improving the lives of home care workers: A partnership of social work and labor. Soc Work 38(5):579-585..

Edling, C. 1980. Anesthetic gases as an occupational hazard. A review. Scand J Work Environ Health 6:85-93.

Ehrengut, W and T Klett. 1981. Rötelnimmunstatus von Schwesternschülerinnen in Hamberger Krankenhäusern im Jahre 1979. Monatsschrift Kinderheilkdunde 129:464-466.

Elias, J, D Wylie, A Yassi, and N Tran. 1993. Eliminating worker exposure to ethylene oxide from hospital sterilizers: An evaluation of cost and effectiveness of an isolation system. Appl Occup Environ Hyg 8(8):687-692.

Engels, J, TH Senden, and K Hertog. 1993. Working postures of nurses in nursing homes. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Englade J, E Badet and G Becque. 1994. Vigilance et qualité de sommeil des soignants de nuit. Revue de l’infirmière 17:37-48.

Ernst, E and V Fialka. 1994. Idiopathic low back pain: Present impact, future directions. European Journal of Physical Medicine and Rehabilitation 4:69-72.

Escribà Agüir, V. 1992. Nurses’ attitudes towards shiftwork and quality of life, Scand J Soc Med 20(2):115-118.

Escribà Agüir V, S Pérez, F Bolumar, and F Lert. 1992. Retentissement des horaires de travail sur le sommeil des infirmiers. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Estryn-Béhar, M. 1990. Les groupes de parole: Une stratégie d’amélioration des relations avec les malades. Le concours médical 112(8):713-717.

—. 1991. Guide des risques professionnels du personnel des services de soins. Paris: Editions Lamarre.

Estryn-Béhar, M and N Bonnet. 1992. Le travail de nuit à l’hôpital. Quelques constats à mieux prendre en compte. Arch Mal Prof 54(8):709-719.

Estryn-Béhar, M and F Fonchain. 1986. Les troubles du sommeil du personnel hospitalier effectuant un travail de nuit en continu. Arch Mal Prof 47(3):167-172;47(4):241.

Estryn-Béhar, M and JP Fouillot. 1990a. Etude de la charge physique du personnel soignant, Documents pour le médecin du travail. INRS: 27-33.

—. 1990b. Etude de la charge mentale et approche de la charge psychique du personnel soignant. Analyse du travail des infirmières et aides-soignantes dans 10 services de soins. Documents pour le médecin du travail INRS 42:131-144.

Estryn-Béhar, M and C Hakim-Serfaty. 1990. Organisation de l’espace hospitalier. Techn hosp 542:55-63.

Estryn-Béhar, M and G Milanini. 1992. Concevoir les espaces de travail en services de soins. Technique Hospitalière 557:23-27.

Estryn-Béhar, M and H Poinsignon. 1989. Travailler à l’hopital. Paris: Berger Levrault.

Estryn-Béhar, M, C Gadbois, and E Vaichere. 1978. Effets du travail de nuit en équipes fixes sur une population féminine. Résultats d’une enquête dans le secteur hospitalier. Arch Mal Prof 39(9):531-535.

Estryn-Béhar, M, C Gadbois, E Peigné, A Masson, and V Le Gall. 1989b. Impact of nightshifts on male and female hospital staff, in Shiftwork: Health and Performance, edited by G Costa, G Cesana, K Kogi, and A Wedderburn. Proceedings of the International Symposium on Night and Shift Work. Frankfurt: Peter Lang.

Estryn-Béhar, M, M Kaminski, and E Peigné. 1990. Strenuous working conditions and musculoskeletal disorders among female hospital workers. Int Arch Occup Environ Health 62:47-57.

Estryn-Béhar, M, M Kaminski, M Franc, S Fermand, and F Gerstle F. 1978. Grossesse er conditions de travail en milieu hospitalier. Revue franç gynec 73(10) 625-631.

Estryn-Béhar, M, M Kaminski, E Peigné, N Bonnet, E Vaichère, C Gozlan, S Azoulay, and M Giorgi. 1990. Stress at work and mental health status. Br J Ind Med 47:20-28.

Estryn-Béhar, M, B Kapitaniak, MC Paoli, E Peigné, and A Masson. 1992. Aptitude for physical exercise in a population of female hospital workers. Int Arch Occup Environ Health 64:131-139.

Estryn Béhar, M, G Milanini, T Bitot, M Baudet, and MC Rostaing. 1994. La sectorisation des soins: Une organisation, un espace. Gestion hospitalière 338:552-569.

Estryn-Béhar, M, G Milanini, MM Cantel, P Poirier, P Abriou, and the ICU’s study group. 1995a. Interest of participative ergonomic methodology to improve an intensive care unit. In Occupational Health for Health Care Workers, 2nd edition, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

—. 1995b. Participative ergonomic methodology for the new fitting out of a cardiologic intensive care unit. In Occupational Health for Health Care Workers, 2nd edition, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Estryn-Béhar, M, E Peigné, A Masson, C Girier-Desportes, JJ Guay, D Saurel, JC Pichenot, and J Cavaré. 1989a. Les femmes travaillant à l’hôpital aux différents horaires, qui sont-elles? Que décrivent-elles comme conditions de travail? Que souhaitent-elles? Arch Mal Prof 50(6):622-628.

Falk, SA and NF Woods. 1973. Hospital noise-levels and potential health hazards, New England J Med 289:774-781.

Fanger, PO. 1973. Assessment of man’s thermal comfort in practice. Br J Ind Med 30:313-324.

—. 1992. Sensory characterization of air quality and pollution sources. In Chemical, Microbiological, Health and Comfort Aspects of Indoor Air Quality—State of the Art in SBS, edited by H Knoppel and P Wolkoff. Dordrecht, NL: Kluwer Academic Publishers.

Favrot-Laurens. 1992. Advanced technologies and work organization of hospital teams. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

—. 1992. Sensory characterization of air quality and pollution sources. In Chemical, Microbiological, Health and Comfort Aspects of Indoor Air Quality—State of the Art in Sick Building Syndrome, edited by H Koppel and P Wolkoff. Brussels and Luxembourg: EEC.

Ferstandig, LL. 1978. Trace concentrations of anesthetic gases: A critical review of their disease potential. Anesth Analg 57:328-345.

Finley, GA and AJ Cohen. 1991. Percieved urgency and the anaesthetist: Responses to common operating room monitor alarms. Can J Anaesth 38(8):958-964

Ford, CV and DK Wentz. 1984. The internship year: A study of sleep, mood states, and psychophysiologic parameters. South Med J 77:1435-1442.

Friedman, RC, DS Kornfeld, and TJ Bigger. 1971. Psychological problems associated with sleep deprivation in interns. Journal of Medical Education 48:436-441.

Friele, RD and JJ Knibbe. 1993. Monitoring the barriers with the use of patient lifts in home care as perceived by nursing personnel. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. LandsbergLech: Ecomed Verlag.

Gadbois, CH. 1981. Aides-soignantes et infirmières de nuit. In Conditions de travail et vie quotidienne. Montrougs: Agence Nationale pour l’Amélioration des Conditions de Travail.

Gadbois, C, P Bourgeois, MM Goeh-Akue-Gad, J Guillaume, and MA Urbain. 1992. Contraintes temporelles et structure de l’espace dans le processus de travail des équipes de soins. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Games, WP, and W Tatton-Braen. 1987. Hospitals Design and Development. London: Architectural Press.

Gardner, ER and RC Hall. 1981. The professional stress syndrome. Psychosomatics 22:672-680.

Gaube, J, H Feucht, R Laufs, D Polywka, E Fingscheidt, and HE Müller. 1993. Hepatitis A, B und C als desmoterische Infecktionen. Gessundheitwesen und Desinfextion 55:246-249.

Gerberding, JL. N.d. Open trial of Zidovudine Postexposure-chemoprophylaxis in Health Care Workers with Occupational Exposures to Human Immunodeficiency Virus. Skript SFGH.

—. 1995. Management of occupational exposures to blood-borne viruses. New Engl J Med 332:444-451.

Ginesta, J. 1989. Gases anestésicos. In Riesgos del Trabajo del Personal Sanitario, edited by JJ Gestal. Madrid: Editorial Interamericana McGraw-Hill.

Gold, DR, S Rogacz, N Bock, TD Tosteson, TM Baum, FE Speizer, and CA Czeiler. 1992. Rotating shift work, sleep and accidents related to sleepiness in hospital nurses. Am J Public Health 82(7):1011-1014.

Goldman, LI, MT McDonough, and GP Rosemond. 1972. Stresses affecting surgical performance and learning: Correlation of heart rate, electrocardiogram, and operation simultaneously recorded on videotapes. J Surg Res 12:83-86.

Graham, C, C Hawkins, and W Blau. 1983. Innovative social work practice in health care: Stress management. In Social Work in a Turbulent World, edited by M Dinerman. Washington, DC: National Association of Social Workers.

Green, A. 1992. How nurses can ensure the sounds patients hear have a positive rather than negative effect upon recovery and quality of life. Intensive & Critical Care Nursing Journal 8(4):245-248.

Griffin, WV. 1995. Social worker and agency safety. In Encyclopaedia of Social Work, 19th edition. Washington, DC: National Association of Social Workers.

Grob, PJ. 1987. Cluster of hepatitis B transmission by a physician. Lancet 339:1218-1220.

Guardino, X and MG Rosell. 1985. Exposicion laboral a gases anestésicos. In Notas Técnicas de Prevención. No. 141. Barcelona: INSHT.

—. 1992. Exposure at work to anesthetic gases. A controlled risk? Janus 12:8-10.

—. 1995. Exposure monitoring to anesthetic gases. In Occupational Health for Health Care Workers, edited by M Hagburg, F Hoffmann, U Stössel, and G Westlander. Solna: National Institute of Occupational Health.

Hagberg, M, F Hofmann, U Stössel, and G Westlander (eds.). 1993. Occupational Health for Health Care Workers. Landsberg/Lech: Ecomed Verlag.

Hagberg, M, F Hofmann, U Stössel, and G Westlander (eds.). 1995. Occupational Health for Health Care Workers. Singapore: International Commission on Occupational Health.

Haigh, R. 1992. The application of ergonomics to the design of workplace in health care buildings in the U.K. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Halm, MA and MA Alpen, 1993. The impact of technology on patient and families. Nursing Clinics of North America 28(2):443-457.

Harber, P, L Pena, and P Hsu. 1994. Personal history, training, and worksite as predictors of back pain of nurses. Am J Ind Med 25:519-526.

Hasselhorn, HM. 1994. Antiretrovirale prophylaxe nach kontakt mit HIV-jontaminierten. In Flüssigkeiten in Infektiologie, edited by F Hofmann. Landsberg/Lech: Ecomed Verlag.

Hasselhorn, HM and E Seidler.1993. Terminal care in Sweden—New aspects of the professional care of dying. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel U, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Heptonstall, J, K Porter, and N Gill. 1993. Occupational Transmission of HIV: Summary of Published Reports. London: Communicable Disease Surveillance Centre AIDS Centre.

Hesse, A, Lacher A, HU Koch, J Kublosch, V Ghane, and KF Peters. 1996. Update on the latex allergy topic. Hauzarzt 47(11):817-824.

Ho, DD, T Moudgil, and M Alam. 1989. Quantitation of human immunodeficiency virus type 1 in the blood of infected persons. New Engl J Med 321:1621-1625.

Hodge, B and JF Thompson. 1990. Noise pollution in the operating theatre. Lancet 335:891-894.

Hofmann, F and H Berthold. 1989. Zur Hepatitis-B-Gefährdung des Krankenhauspersonals-Möglichkeiten der prae-und postexpositionellen Prophylaxe. Medizinische Welt 40:1294-1301.

Hofmann, F and U Stössel. 1995. Environmental health in the health care professions: Biological, physical, psychic, and social health hazards. Reviews on Environmental Health 11:41-55.

Hofmann, F, H Berthold, and G Wehrle. 1992. Immunity to hepatitis A in hospital personnel. Eur J Clin Microbiol Infect Dis 11(12):1195.

Hofmann, F, U Stössel, and J Klima. 1994. Low back pain in nurses (I). European Journal of Physical and Medical Rehabilitation 4:94-99.

Hofmann, F, B Sydow, and M Michaelis. 1994a. Mumps—berufliche Gefährdung und Aspekte der epidemiologischen Entwicklung. Gessundheitwesen und Desinfextion 56:453-455.

—. 1994b. Zur epidemiologischen Bedeutung der Varizellen. Gessundheitwesen und Desinfextion 56:599-601.

Hofmann, F, G Wehrle, K Berthold, and D Köster. 1992. Hepatitis A as an occupational hazard. Vaccine 10 suppl 1:82-84.

Hofmann, F, U Stössel, M Michaelis, and A Siegel. 1993. Tuberculosis—Occupational risk for health care workers? In Occupational Health for Health Care Workers, edited by M Hagberg. Landsberg/Lech: Ecomed Verlag.

Hofmann, F, M Michaelis, A Siegel, and U Stössel. 1994. Wirbelsäulenerkrankungen im Pflegeberuf. Medizinische Grundlagen und Prävention. Landsberg/Lech: Ecomed Verlag.

Hofmann, F, M Michaelis, M Nübling, and FW Tiller. 1995. European Hepatitis—A Study. Publikation in Vorereitung.

Hofmann, H and C Kunz. 1990. Low risk of health care workers for infection with hepatitis-C virus. Infection 18:286-288.

Holbrook, TL, K Grazier, JL Kelsey, and RN Stauffer. 1984. The Frequency of Occurrence, Impact, and Cost of Selected Musculoskeletal Conditions in the United States. Park Ridge, Il: American Academy of Orthopedic Surgeons.

Hollinger, FB. 1990. Hepatitis B virus. In Virology, edited by BN Fiedles and DM Knipe. New York: Raven Press.

Hopps, J and P Collins. 1995. Social work profession overview. In Encyclopedia of Social Work, 19th edition. Washington, DC: National Association of Social Workers.

Hubacova, L, I Borsky, and F Strelka. 1992. Work physiology problems of nurses working in inpatients departments. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Hunt, LW, AF Fransway, CE Reed, LK Miller, RT Jones, MC Swanson, and JW Yunginger. 1995. An epidemic of occupational allergy to latex involving health care workers. J Occup Environ Med 37(10):1204-1209.

Jacobson, SF and HK MacGrath. 1983. Nurses under Stress. New York: John Wiley & Sons.

Jacques, CHM, MS Lynch and JS Samkoff. 1990. The effects of sleep loss on cognitive performance of resident physicians. J Fam Pract 30:223-229.

Jagger, J, EH Hunt, J Brand-Elnagger, and RD Pearson. 1988. Rates of needle-stick injury caused by various devices in a university hospital. New Engl J Med 319:284-288.

Johnson, JA, RM Buchan, and J S Reif. 1987. Effect of waste anesthetic gas and vapor exposure on reproductive outcome in veterinary personnel. Am Ind Hyg Assoc J 48(1):62-66.

Jonasson, G, JO Holm, and J Leegard. Rubber allergy: An increasing health problem? Tuidsskr Nor Laegeforen 113(11):1366-1367.

Kandolin, I. 1993. Burnout of female and male nurses in shiftwork. Ergonomics 36(1/3):141-147.

Kaplan, RM and RA Deyo. 1988. Back pain in health care workers. In Back Pain in Workers, edited by RA Deyo. Philadelphia, PA: Hanley & Belfus.

Katz, R. 1983. Causes of death among nurses. Occup Med 45:760-762.

Kempe, P, M Sauter and I Lindner. 1992. Special characteristics of nurses for the aged who made use of a training program aimed to reduce burn-out symptoms and first results on treatment outcome. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Kerr, JH. 1985. Warning devices. Br J Anaesth 57:696-708.

Kestin, IG, RB Miller, and CJ Lockhart. 1988. Auditory alarms during anesthesia monitoring. Anesthesiology 69(1):106-109.

Kinloch-de-los, S, BJ Hirschel, B Hoen, DA Cooper, B Tindall, A Carr, H Sauret, N Clumeck, A Lazzarin, and E Mathiesen. 1995. A controlled trial of Zidovudine in primary human immunodeficiency virus infection. New Engl J Med 333:408-413.

Kivimäki, M and K Lindström. 1995. The crucial role of the head nurse in a hospital ward. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Klaber Moffet, JA, SM Chase, I Portek, and JR Ennis. 1986. A controlled study to evaluate the efectiveness of the back pain school in the relief of chronic low back pain. Spine 11:120-122.

Kleczkowski, BM, C Montoya-Aguilar, and NO Nilsson. 1985. Approaches to Planning and Design of Health Care Facilities in Developing Areas. Vol. 5. Geneva: WHO.

Klein, BR and AJ Platt. 1989. Health Care Facility Planning and Construction. New York: Van Nostrand Reinhold.

Kelin, R, K Freemann, P Taylor, C Stevens. 1991. Occupational risk for hepatits C virus infection among New York City dentists. Lancet 338:1539-1542.

Kraus, H. 1970. Clinical Treatment of Back and Neck Pain. New York: McGraw-Hill.

Kujala, VM and KE Reilula. 1995. Glove-induced dermal and respiratory symptoms among health care workers in one Finnish hospital. Am J Ind Med 28(1):89-98.

Kurumatani, N, S Koda, S Nakagiri, K Sakai, Y Saito, H Aoyama, M Dejima, and T Moriyama. 1994. The effects of frequently rotating shiftwork on sleep and the family life of hospital nurses. Ergonomics 37:995-1007.

Lagerlöf, E and E Broberg. 1989. Occupational injuries and diseases. In Occupational Hazards in the Health Professions, edited by DK Brune and C Edling. Boca Raton, FL: CRC Press.

Lahaye, D, P Jacques, G Moens, and B Viaene. 1993. The registration of medical data obtained by preventive medical examinations on health care workers. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, F, U Stössel and G Westlander. Landsberg/Lech: Ecomed Verlag.

Lampher, BP, CC Linneman, CG Cannon, MM DeRonde, L Pendy, and LM Kerley. 1994. Hepatitis C virus infection in health care workers: Risk of exposure and infection. Infect Control Hosp Epidemiol 15:745-750.

Landau, C, S Hall, SA Wartman, and MB Macko. 1986. Stress in social and family relationships during medical residency. Journal of Medical Education 61:654-660.

Landau, K. 1992. Psycho-physical strain and the burn-out phenomen amongst health care professionals. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Landewe, MBM and HT Schröer. 1993. Development of a new, integrated patient transfer training program—Primary prevention of low back pain. In Occupational Health for Health Care Workers, editeb by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Lange, M. 1931. Die Muskelhärten (Myogelosen). Munich: JF Lehman Verlag.

Lange, W and KN Masihi. 1986. Durchseuchung mit Hepatitis-A- und B-Virus bei medizinischem Personal. Bundesgesundheitsol 29;183-87.

Lee, KA. 1992. Self-reported sleep disturbances in employed women. Sleep15(6):493-498.

Lempereur, JJ. 1992. Prévention des dorso-lombalgies. Influence du vêtement de travail sur le comportement gestuel. Spécifications ergonomiques. Cah Kinésither 156,:4.

Leppanen, RA and MA Olkinuora. 1987. Psychological stress experienced by health care personnel. Scand J Work Environ Health 13:1-8.

Lert, F, MJ Marne, and A Gueguen. 1993. Evolution des conditions de travail des infirmières des hôpitaux publics de 1980 à 1990. Revue de l’Epidémiologie et de santé publique 41:16-29.

Leslie, PJ, JA Williams, C McKenna, G Smith and RC Heading. 1990. Hours, volume, and type of work of preregistration house officers. Brit Med J 300:1038-1041.

Lettau, LA, HJ Alfred, RH Glew, HA Fields, MJ Alter, R Meyer, SC Hadler, and JE Maynard. 1986. Nosocomial transmission of delta hepatitis. Ann Intern Med 104:631-635.

Levin, H. 1992. Healthy buildings—Where do we stand, where do we go? In Chemical, Microbiological, Health and Comfort Aspects of Indoor Air Quality: State of the Art in Sick Building Syndrome, edited by H Knoppel and P Wolkoff. Brussels and Luxembourg: EEC.

Lewittes, LR and VW Marshall. 1989. Fatigue and concerns about quality of care among Ontario interns and residents. Can Med Assoc J 140:21-24.

Lewy, R. 1990. Employees at Risk: Protection and Health of Health Care Workers. New York: Van Nostrand Reinhold.

Lindström, A and M Zachrisson. 1973. Ryggbesvär och arbetssoförmaga Ryyggskolan. Ett Försok till mer rationeli fysikalist terapi. Socialmet T 7:419-422.

Lippert. 1971. Travel in nursing units. Human Factors 13(3):269-282.

Ljungberg, AS, A Kilbom, and MH Goran. 1989. Occupational lifting by nursing aides and warehouse workers. Ergonomics 32:59-78.

Llewelyn-Davies, R and J Wecks. 1979. In-patient areas. In Approaches to Planning and Design of Health Care Facilities in Developing Areas, edited by BM Kleczkowski and R Piboleau. Geneva: WHO.

Loeb, RG, BR Jones, KH Behrman, and RJ Leonard. 1990. Anesthetists cannot identify audible alarms. Anesthesiology 73(3A):538.

Lotas, MJ. 1992. Effects of light and sound in the neonatal intensive care unit environment on the low-birth-weight infant. NAACOGS Clinical Issues in Perinatal & Womens Health Nursing 3(1):34-44.

Lurie, HE, B Rank, C Parenti, T Wooley, and W Snoke. 1989. How do house officers spend their nights? A time study of internal medicine house staff on call. New Engl J Med 320:1673-1677.

Luttman, A, M Jäger, J Sökeland, and W Laurig. 1996. Electromyographical study on surgeons in urology II. Determination of muscular fatigue. Ergonomics 39(2):298-313.

Makino, S. 1995. Health problems in health care workers in Japan. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsbeg/Lech: Ecomed Verlag.

Malchaire, JB. 1992. Analysis of the work load of nurses. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Manuaba, A. 1992. Social-cultural approach is a must in designing hospital in developing countries, Indonesia as a case study. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Maruna, H. 1990. Zur Hepatitis-B-Durchseuchung in den Berufen des Gesundheits und Fürsorgewesens der Republik Österreichs, Arbeitsmed. Präventivmed. Sozialmed 25:71-75.

Matsuda, A. 1992. Ergonomics approach to nursing care in Japan. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

McCall, T. 1988. The impact of long working hours on resident physicians. New Engl J Med 318(12):775-778.

McCloy, E. 1994. Hepatitis and the EEC Directive. Presented at the 2nd International Conference on Occupational Health for Health Care Workers, Stockholm.

McCormick, RD, MG Meuch, IG Irunk, and DG Maki. 1991. Epidemiology for hospital sharp injuries: A 14-year prospective study in the pre-AIDS and AIDS era. Am J Med 3B:3015-3075.

McCue, JD. 1982. The effects of stresses on physicians and their medical practice. New Engl J Med 306:458-463.

McIntyre, JWR. 1985. Ergonomics: Anaesthetists’ use of auditory alarms in the operating room. Int J Clin Monit Comput 2:47-55

McKinney, PW, MM Horowitz, and RJ Baxtiola. 1989. Susceptibility of hospital-based health care personnel to varicella zoster virus infection. Am J Infect Control 18:26-30.

Melleby, A. 1988. Exercise program for a healthy back. In Diagnosis and Treatment of Muscle Pain. Chicago, IL: Quintessence Books.

Meyer,TJ, SE Eveloff, MS Bauer, WA Schwartz, NS Hill, and PR Millman. 1994. Adverse environmental conditions in the respiratory and medical intensive care unit settings. Chest 105:1211-1216.

Miller, E, J Vurdien, and P Farrington. 1993. Shift age in chickenpox. Lancet 1:341.

Miller, JM. 1982. William Stewart Halsted and the use of the surgical rubber glove. Surgery 92:541-543.

Mitsui, T, K Iwano, K Maskuko, C Yanazaki, H Okamoto, F Tsuda, T Tanaka, and S Mishiros. 1992. Hepatitis C virus infection in medical personnel after needlestick accidents. Hepatology 16:1109-1114.

Modig, B. 1992. Hospital ergonomics in a biopsychosocial perspective. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Momtahan, K, R Hétu, and B Tansley. 1993. Audibility and identification of auditory alarms in the operating room and intensive care unit. Ergonomics 36(10):1159-1176.

Momtahan, KL and BW Tansley. 1989. An ergonomic analysis of the auditory alarm signals in the operating room and recovery room. Presented at the Annual Meeting of the Canadian Acoustical Association, 18 October, Halifax, NS.

Montoliu, MA, V Gonzalez, B Rodriguez, JF Quintana, and L Palenciano.1992. Conditions de travail dans la blanchisserie centrale des grands hôpitaux de Madrid. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Moore, RM, YM Davis, and RG Kaczmarek. 1993. An overview of occupational hazards among veterinarians, with particular reference to pregnant women. Am J Ind Hyg Assoc 54(3):113-120.

Morel, O. 1994. Les agents des services hospitaliers. Vécu et santé au travail. Arch mal prof 54(7):499-508.

Nachemson, AL and GBJ Anderson. 1982. Classification of low back pain. Scand J Work Environ Health 8:134-136.

National Health Service (NHS). 1991a. Design Guide. The Design of Community Hospitals. London: Her Majesty’s Stationery Office.

—. 1991b. Health Building Note 46: General Medical Practice Premises for the Provision of Primary Health Care Service. London: Her Majesty’s Stationery Office.

National Institute for Occupational Safety and Health (NIOSH). 1975. Development and Evaluation of Methods for the Elimination of Waste Anesthetic Gases and Vapors in Hospitals. DHEW (NIOSH) Publication No. 75-137. Cincinnati, OH: NIOSH.

—. 1997a. Control of Occupational Exposure to N2O in the Dentral Operatory. DHEW (NIOSH) Publication No. 77-171. Cincinnati, OH: NIOSH.

—. 1977b. Criteria for a Recommended Standard: Occupational Exposure to Waste Anesthetic Gases and Vapors. DHEW (NIOSH) Publication No. 77-1409. Cincinnati, OH: NIOSH.

—. 1988. Guidelines for Protecting the Safety and Health of Health Care Workers. DHHS (NIOSH) Publication No. 88-119. Cincinnati, OH: NIOSH.

—. 1994. NIOSH Alert: Request for Assistance in Controlling Exposures to Nitrous Oxide during Anesthetic Administration. DHHS (NIOSH) Publication No. 94-100. Cincinnati, OH: NIOSH.

Niu, MT, DS Stein, and SM Schnittmann. 1993. Primary human immunodeficiency virus type 1 infection: Review of pathogenesis and early treatment interventions in human and animal retrovirus infections. J Infect Dis 168:1490-1501.

Noweir, MH and MS al-Jiffry. 1991. Study of noise pollution in Jeddah hospitals. Journal of the Egyptian Public Health Association 66 (3/4):291-303.

Nyman, I and A Knutsson. 1995. Psychosocial wellbeing and sleep quality in hospital night and day workers. In Occuaptional Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Objectif Prévention No spécial. 1994. Le lève personne sur rail au plafond: Outil de travail indispensable. Objectif Prévention 17(2):13-39.

O’Carroll, TM. 1986. Survey of alarms in an intensive therapy unit. Anaesthesia 41:742-744.

Occupational Safety and Health Administration (OSHA). 1991. Occupational Exposure to Bloodborne Pathogens: Final Rule. 29 CFR Part 1910.1030. Washington, DC: OSHA.

Oëler, JM. 1993. Developmental care of low birth weight infants. Nursing Clinics of North America 28(2):289-301.

Öhling, P and B Estlund. 1995. Working technique for health care workers. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander G. Landsberg/Lech: Ecomed Verlag.

Ollagnier, E and Lamarche MJ. 1993. Une intervention ergonomique dans un hôpital suisse: Impact sur la santé de l’organisation du personnel et des patients. In Ergonomie et santé, edited by D Ramaciotti and A Bousquet. Actes du XXVIIIe congrès de la SELF. Geneva: SELF.

Ott, C, M Estryn-Béhar, C Blanpain, A Astier, and G Hazebroucq. 1991. Conditionnement du médicament et erreurs de médication. J Pharm Clin 10:61-66.

Patkin, M. 1992. Hospital architecture: An ergonomic debacle. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Payer, L. 1988. Medicine and Culture: The Variety of Treatment in the United States, England, West Germany and France. New York: H. Holt.

Payne, R and J Firth-Cozens (eds.). 1987. Stress in Health Professions. New York: John Wiley & Sons.

—. 1995. Determination of dinitrogen oxide (N2O) in urine as control to anesthetic exposure. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hoffmann, U Stössel, and G Westlander. Solna: National Institute of Occupational Health.

Pelikan, JM. 1993. Improving occupational health for health care workers within the health promoting hospital: Experiences from the Vienna WHO model project “health and hospital”. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Pérez, L, R De Andrés, K. Fitch, and R Najera. 1994. Seroconversiones a VIH tras Sanitarios en Europa. Presented at the 2nd Reunión Nacional sobre el SIDA Cáceres.

Philip, RN, KRT Reinhard, and DB Lackman. 1959. Observations on a mumps epidemic in a “virgin” population. Am J Hyg 69:91-111.

Pottier, M. 1992. Ergonomie à l’hôpital-hospital ergonomics. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Poulton, EC, GM Hunt, A Carpenter, and RS Edwards. 1978. The performance of junior hospital doctors following reduced sleep and long hours of work. Ergonomics 21:279-295.

Pöyhönen, T and M Jokinen. 1980. Stress and Other Occupational Health Problems Affecting Hospital Nurses. Vantaa, Finland: Tutkimuksia.

Raffray, M. 1994. Etude de la charge physique des AS par mesure de la fréquence cardiaque. Objectif soins 26:55-58.

Ramaciotti, D, S Blaire, A Bousquet, E Conne, V Gonik, E Ollagnier, C Zummermann, and L Zoganas. 1990. Processus de régulation des contraintes économiques physiologiques et sociales pour différents groupes de travail en horaires irréguliers et de nuit. Le travail humain 53(3):193-212.

Reuben, DB. 1985. Depressive symptoms in medical house officers: Effects of level of training and work rotation. Arch Intern Med 145:286-288.

Reznick, RK and JR Folse. 1987. Effect of sleep deprivation on the performance of surgical residents. Am J Surg 154:520-52.

Rhoads, JM.1977. Overwork. JAMA 237:2615-2618.

Rodary, C and A Gauvain-Piquard 1993. Stress et épuisement professionnel. Objectif soins 16:26-34.

Roquelaure, Y, A Pottier, and M Pottier. 1992. Approche ergonomique comparative de deux enregistreurs electroencéphalographiques. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Rosell, MG, P Luna, and X Guardino. 1989. Evaluacion y Control de Contaminantes QuPmicos en Hospitales. Technical Document No. 57. Barcelona: INSHT.

Rubin, R, P Orris, SL Lau, DO Hryhorczuk, S Furner, and R Letz. 1991. Neurobehavioral effects of the on-call experience in housestaff physicians. J Occup Med 33:13-18.

Saint-Arnaud, L, S Gingras, R Boulard., M Vezina and H Lee-Gosselin. 1992. Les symptômes psychologiques en milieu hospitalier. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Samkoff, JS, CHM Jacques. 1991. A review of studies concerning effects of sleep deprivation and fatigue on residents’ performance. Acad Med 66:687-693.

Sartori, M, G La Terra, M Aglietta, A Manzin, C Navino, and G Verzetti. 1993. Transmission of hepatitis C via blood splash into conjunctiva. Scand J Infect Dis 25:270-271.

Saurel, D. 1993. CHSCT Central, Enquete “Rachialgies” Résultats. Paris: Assistance Publique-Höpitaux de Paris, Direction du personnel et des relations sociales.

Saurel-Cubizolles, MJ, M Hay, and M Estryn-Béhar. 1994. Work in operating rooms and pregnancy outcome among nurses. Int Arch Occup Environ Health 66:235-241.

Saurel-Cubizolles, MJ, MKaminski, J Llhado-Arkhipoff, C Du Mazaubrum, M Estryn-Behar, C Berthier, M Mouchet, and C Kelfa. 1985. Pregnancy and its outcome among hospital personnel according to occupation and working condition. Journal of Epidemiology and Community Health 39:129-134.

Schröer, CAP, L De Witte, and H Philipsen. 1993. Effects of shift work on quality of sleep, health complaints and medical consumption of female nurses. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Senevirane, SR, De A and DN Fernando. 1994. Influence of work on pregnancy outcome. Int J Gynecol Obstet VOL: 35-40.

Shapiro, ET, H Pinsker and JH Shale. 1975. The mentally ill physician as practitioner. JAMA 232(7):725-727.

Shapiro, RA and T Berland. 1972. Noise in the operating room. New Engl J Med 287(24):1236-1238.

Shindo, E. 1992. The present condition of nursing ergonomics in Japan. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Siegel, A, M Michaelis, F Hofmann, U Stössel, and W Peinecke. 1993. Use and acceptance of lifting aids in hospitals and geriatric homes. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Smith, MJ, MJ Colligan, IJ Frocki, and DL Tasto. 1979. Occupational injury rates among nurses as a function of shift schedule. Journal of Safety Research 11(4):181-187.

Smith-Coggins, R, MR Rosekind, S Hurd, and KR Buccino. 1994. Relationship of day versus night sleep to physician performance and mood. Ann Emerg Med 24:928-934.

Snook, SH. 1988a. Approaches to the control of back pain in industry. In Back Pain in Workers, edited by RA Deyo. Philadelphia: Hanley & Belfus.

—. 1988b. The costs of back pain in industry. In Back Pain in Workers, edited by RA Deyo. Philadelphia: Hanley & Belfus.

South, MA, JL Sever, and L Teratogen. 1985. Update: The congenital rubella syndrome. Teratology 31:297-392.

Spence, AA. 1987. Environmental pollution by inhalation anaesthetics. Br J Anaesth 59:96-103.

Stellman, JM. 1976. Women’s Work, Women’s Health: Myths and Realities. New York: Pantheon.

Steppacher, RC and JS Mausner. 1974. Suicide in male and female physicians. JAMA 228(3):323-328.

Sterling, DA. 1994. Overview of health and safety in the health care environment. In Essentials of Modern Hospital Safety, edited by W Charney. Boca Raton, FL: Lewis Publishers.

Stoklov, M, P Trouiller, P Stieglitz, Y Lamalle, F Vincent, A Perdrix, C Marka, R de Gaudemaris, JM Mallion, and J Faure. 1983. L’exposition aux gaz anethésiques: Risques et prévention. Sem Hôs 58(29/39):2081-2087.

Storer, JS, HH Floyd, WL Gill, CW Giusti, and H Ginsberg. 1989. Effects of sleep deprivation on cognitive ability and skills of pediatrics residents. Acad Med 64:29-32.

Stubbs, DA, PW Buckle, and PM Hudson. 1983. Back pain in the nursing profession; I Epidemiology and pilot methodology. Ergonomics 26:755-765.

Sundström-Frisk C and M Hellström.1995. The risk of making treatment errors, an occupational stressor. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Swann-D’Emilia, B, JCH Chu, and J Daywalt. 1990. Misadministration of prescribed radiation dose. Medical Dosimetry 15:185-191.

Sydow, B and F Hofmann. 1994. Unpublished results.

Tannenbaum, TN and RJ Goldberg. 1985. Exposure to anaesthetic gases and reproductive outcome: A review of epidemiologic literature. J Occup Med 27:659-671.

Teyssier-Cotte, C, M Rocher, and P Mereau. 1987. Les lits dans les établissements de soins. Documents pour le médecin du travail. INRS 29:27-34.

Theorell, T. 1989. The psychosocial working environment. In Occupational Hazards in the Health Professions, edited by DK Brune and C Edling. Boca Raton, FL: CRC Press.

Theorell T. 1993. On the psychosocial environment in care. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech : Ecomed Verlag.

Tintori, R and M Estryn-Béhar. 1994. Communication: Où, quand, comment? Critères ergonomiques pour améliorer la communication dans les services de soins. Gestions Hospitalières 338:553-561.

Tintori, R, M Estryn-Behar, J De Fremont, T Besse, P Jacquenot, A Le Vot, and B Kapitaniak. 1994. Evaluation des lits à hauteur variable. Une démarche de recherche en soins infirmiers. Gestions Hospitalières 332:31-37.

Tokars, JI, R Marcus, DH Culver, CA Schable, PS McKibben, CL Bandea, and DM Bell. 1993. Surveillance of HIV infection and zidovudine use among health care workers after occupational exposure to HIV-infected blood. Ann Intern Med 118:913-919.

Toomingas, A. 1993. The health situation among Swedish health care workers. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Topf, M. 1992. Effects of personal control over hospital noise on sleep. Research in Nursing & Health 15(1):19-28.

Tornquist, A and P Ullmark. 1992. Corporate Space and Architecture, Actors and Procedures. Paris: Ministère de l’équipement du logement et des transports.

Townsend, M. 1994. Just a glove? Br J Theatre Nurs 4(5):7,9-10.

Tran, N, J Elias, T Rosenber, D Wylie, D Gaborieau, and A Yassi. 1994. Evaluation of waste anesthetic gases, monitoring strategies and corelations between nitrous oxide levels and health symptoms. Am Ind Hyg Assoc J 55(1):36-42.

Turner, AG, CH King, and G Craddock. 1975. Measuring and reducing noise. Noise profile of hospital shows that even “quiet” areas are too noisy. Hospital JAHA 49:85-89.

US Preventive Services Task Force. 1989. Guide to Clinical Preventive Services: An Assessment of the Effectiveness of 169 interventions. Baltimore: Williams & Wilkins.

Vaillant, GE, NC Sorbowale, and C McArthur. 1972. Some psychologic vulnerabilities of physicians. New Engl J Med 287:372-375.

Vaisman, AI. 1967. Working conditions in surgery and their effects on the health of anesthesiologists. Eskp Khir Anesteziol 12:44-49.

Valentino, M, MA Pizzichini, F Monaco, and M Governa. 1994. Latex-induced asthma in four healthcare workers in a regional hospital. Occup Med (Oxf) 44(3):161-164.

Valko, RJ and PJ Clayton. 1975. Depression in the internships. Dis Nerv Syst 36:26-29.

Van Damme, P and GA Tormanns. 1993. European risk model. In Proceedings of the European Conference on Hepatitis B as an Occupatioonal Hazard. 10-12.

Van Damme, P, R Vranckx, A Safary, FE Andre, and A Mehevs. 1989. Protective efficacy of a recombinant deoxyribonucleic acid hepatitis B vaccine in institutionalized mentally handicapped clients. Am J Med 87(3A):265-295.

Van der Star, A and M Voogd. 1992. User participation in the design and evaluation of a new model hospital bed. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Van Deursen, CGL, CAM Mul, PGW Smulders and CR De Winter. 1993. Health and working situation of day nurses compared with a matched group of nurses on rotating shift work. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Van Hogdalem, H. 1990. Design guidelines for architects and users. In Building for People in Hospitals, Workers and Consumers. Luxembourg: European Foundation for the Improvement of Living and Working Conditions.

Van Wagoner, R and N Maguire. 1977. A study of hearing loss among employees in a large urban hospital. Canadian Journal of Public Health 68:511-512.

Verhaegen, P, R Cober, DE Smedt, J Dirkx, J Kerstens, D Ryvers, and P Van Daele. 1987. The adaptation of night nurses to different work schedules. Ergonomics 30(9):1301-1309.

Villeneuve, J. 1992. Une demarche d’ergonomie participative dans le secteur hôspitalier. In Ergonomie à l’hôpital (Hospital ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

—. 1994. PARC: Des fondations solides pour un projet de rénovation ou de construction Objectif prévention (Montreal) 17(5):14-16.

Wade, JG and WC Stevens. 1981. Isoflurane: An ansaesthetic for the eighties? Anesth Analg 60(9):666-682.

Wahlen, L. 1992. Noise in the intensive care setting. Canadian Critical Care Nursing Journal, 8/9(4/1):9-10.

Walz, T, G Askerooth, and M Lynch. 1983. The new upside-down welfare state. In Social Work in a Turbulent World, edited by M Dinerman. Washington, DC: National Association of Social Workers.

Wands, SE and A Yassi. 1993. Modernization of a laundry processing plant: Is it really an improvement? Appl Ergon 24(6):387-396.

Weido, AJ and TC Sim. 1995. The burgeoning problem of latex sensitivity. Surgical gloves are only the beginning. Postgrad Med 98(3):173-174,179-182,184.

Wiesel, SW, HL Feffer, and RH Rothmann. 1985. Industrial Low Back Pain. Charlottesville,VA: Michie.

Wigaeus Hjelm, E, M Hagberg, and S Hellstrom. 1993. Prevention of musculoskeletal disorders in nursing aides by physical training. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Landsberg/Lech: Ecomed Verlag.

Wigand, R and Y Grenner. 1988. Personaluntersuchungen auf Immunität gegen Masern, Varizellen und Röteln, Saarländ. Ärztebl 41:479-480.

Wilkinson, RT, PD Tyler and CA Varey. 1975. Duty hours of young hospital doctors: Effects on the quality of work. J Occup Psychol 48:219-229.

Willet, KM. 1991. Noise-induced hearing loss in orthopaedic staff. J Bone Joint Surg 73:113-115.

Williams, M and JD Murphy. 1991. Noise in critical care units: A quality assurance approach. Journal of Nursing Care Quality 6(1):53-59.

World Health Organization (WHO). 1990. Guidelines on AIDS and First Aid in the Workplace. WHO AIDS Series No. 7. Geneva: WHO.

—. 1991. Biosafety Guidelines for Diagnostic and Research Laboratories Working with HIV. WHO AIDS Series No. 9. Geneva: WHO.

—. 1995. Weekly Epidemiological Report (13 January).

Wugofski, L. 1995. Occupational accident in health care workers—Epidemiology and prevention. In Occupational Health for Health Care Workers, edited by M Hagberg, F Hofmann, U Stössel, and G Westlander. Singapore: International Commission on Occupational Health.

Yassi, A. 1994. Assault and abuse of health care workers in a large teaching hospital. Can Med Assoc J 151(9):1273-1279.

Yassi, A and M McGill. 1991. Determinants of blood and body fluid exposure in a large teaching hospital: Hazards of the intermittent intravenous procedure. American Journal of Infection Control 19(3):129-135.

—. 1995. Efficacy and cost-effectiveness of a needleless intravenous access system. American Journal of Infection Control 22(2):57-64.

Yassi, A, J Gaborieau, J Elias, and D Willie. 1992. Identification and control of hazardous noise levels in a hospital complex. In Ergonomie à l’hôpital (Hospital Ergonomics), edited by M Estryn-Béhar, C Gadbois, and M Pottier. International Symposium Paris 1991. Toulouse: Editions Octares.

Yassi, A, D Gaborieau, I Gi